Special Methods of Examination.

After having completed a general examination of a lame animal—obtaining the history of the case, noting its temperament, type, size, conformation, position assumed while at repose, swellings or enlargements if present, causing the subject to move to note the degree and character of lameness manifested; palpating and manipulating the parts affected to acquire a fairly definite notion of the nature of an inflammation or to recognize crepitation it becomes necessary in some cases to employ peculiar means of examination in singular instances. This may be done by making use of cocain in solution for the production of local anesthesia as in lameness of the phalanges. Such means are not, in themselves, dependable but are valuable when used in conjunction with all other available and practical methods.

Trial use of various shoes in order to shift the weight from one part of the foot to another or to cause an animal to "break over" in a different manner so that the gait may be changed, constitutes a special test procedure. The use of hoof testers or of a hammer to note the degree or presence of supersensitiveness is another means that is of practical service. No examination, in any case of lameness, is complete without having removed the shoe and scrutinized the solar surface of the foot.



Diagnosis by exclusion, finally, is resorted to, and, as in any other case where the recognition of cause is difficult, exclusion of the existence of conditions,—one at a time, by an analysis of symptoms—generally enables the practictioner to eliminate all but the disturbing element.

FOOTNOTES:

[Footnote 4: By stride is meant the distance between two successive imprints of the same foot. The term is not used in this work as being synonymous with step.]



SECTION III.

LAMENESS IN THE FORE LEG.

Anatomo-Physiological Review of parts of the Fore Leg.

For supporting weight, whether the subject is at rest or in motion, the bony column of the leg, together with attached ligaments, tendons and muscles, is wonderfully well adapted by nature for the function which they perform. The several bones which go to make up the supportive portion of the leg, are so joined at their points of articulation, that a minimum degree of strain is put upon each attachment.

The upper third of the scapula, with its cartilage of prolongation, is sufficiently broad and flattened that it fits snugly against the thorax without necessity for a complicated method of attachment—the clavicle being absent, attachment is muscular.

Smith[5] has very aptly stated that:

"It seems quite legitimate to regard the muscular union between the thorax and forelimb as a joint. There are no bones resting on each other, no synovia; but where the scapula has its largest range of movement there is a remarkable amount of areolar tissue, which renders movement easy. The whole central area beneath the scapula and humerus not occupied by muscular attachment, is filled with this easy-moving, apparently gaseously distended, crepitant, areolar tissue over which the fore legs glide on the chest wall as freely as if the parts were a large, well lubricated joint."

The scapulohumeral articulation (shoulder joint) is an enarthrodial (ball and socket) joint but because of its being held more or less firmly against the thoracic wall by muscular and tendinous attachment, and because a part of this attachment affords a means of support for the body itself, there is no need for binding ligaments and movement is possible in all directions even though restricted as to extent.



Undue extension, (by extension is meant such movement as will cause the long axis of two articulating bones to assume a position which approaches or forms a straight line—opposite to flexion), of the scapulohumeral joint is impossible while weight is borne, because of the normally flexed position of the humerus on the scapula; whereas flexion, beyond desirable limits, is inhibited by the biceps brachii (flexor brachii or coracoradialis) muscle.

The distal end of the humerus, however, articulating with the radius and ulna in a fashion that no support is lent by any sort of contact with the body, is a ginglymus (hinge) joint and lateral motion, because of the long transverse diameter of its articular portions, is easily prevented by the medial and lateral ligaments (internal and external ligaments). Flexion of this, the humeroradioulnar joint (elbow), is restrained by the triceps brachii and extension is checked by the biceps brachii (flexor brachii).

The carpal joint (erroneously called the knee joint), is composed of the several carpal bones which interarticulate and, when taken as a group, serve as a means of attachment and articulation for the radius and metacarpal bones.

The transverse diameter of this joint is long, thus giving it contacting surfaces that are sufficiently extensive to minimize the strain upon the mesial and lateral ligaments (internal and external lateral common ligaments). Motion is that of flexion and extension; slight rotation is possible when the position is that of flexion. While supporting weight the carpus is fixed in position by a slight dorsal flexion, but undue dorsal flexion is prevented by the flexor muscles and tendons and volar-carpal or annular ligament, together with the superior check ligament.

The metacarpophalangeal articulation (fetlock joint), is a hinge joint and its articular surfaces contact one another, with respect to their having a long bearing surface from side to side, as do all ginglymus (hinge) joints. Two common lateral ligaments bind the bones together. While bearing weight, there is assumed a position of slight dorsal flexion, undue flexion being checked by the inhibitory apparatus of the joint—check ligaments, and their tendons and the suspensory ligament. The inhibitory apparatus of the fetlock joint is materially reinforced by the proximal sesamoid bones. Situated as they are, between the bifurcating portions of the suspensory ligament and the posterior part of the distal end of the metacarpus—with which they articulate—the sesamoid bones serve to change the course of the branches of the suspensory ligament in a manner that they give firm support to this joint. Volar flexion is limited by the extensors of the phalanges.



The first phalanx (os suffraginis) normally sets at an angle of about 50 to 55 degrees from a horizontal plane while weight is being supported. Its distal end articulates with the second or median phalanx (os corona) and forms the proximal interphalangeal (pastern or suffraginocoronary) joint. This also, is a ginglymus joint, having but slight lateral motion, and that only when it is in a state of flexion. A rather broad articular surface—from side to side—exists here, lessening the strain on the collateral ligaments somewhat. Dorsal flexion is checked by the flexor tendons and dorsal ligaments. Volar flexion is restrained by the extensor tendons.

The distal end of the second phalanx (os corona) has but slight lateral motion and this is manifested principally when it is in a state of volar flexion. Undue dorsal flexion is prevented by the deep flexor tendon (perforans) and volar flexion is inhibited by the extensor of the digit (extensor pedis). Thus it is seen, that when the leg is a weight-bearing member, weight is supported by the bony framework whose constituent parts are joined together by ligaments and tendons and each one of the several bones articulates in such manner that the joint is locked. The articular parts of bones rest upon or against an inhibitory apparatus, and are slightly flexed, as in the carpus, or considerably flexed such as in the fetlock joint when weight is being supported. In the first instance, for example, the flexors of the carpus and the superior check ligament assisted by the flexors of the phalanges constitute the inhibitory apparatus.

It will be noted that provision for weight bearing is so arranged that muscular energy is not required except in the matter of suspension of the body between the scapulae and here tonic impulses only are necessary to maintain an equilibrium[6], yet in every instance where weight is not supported by bones, inelastic ligaments or tendinous structures relieve the musculature of this constant strain. This explains the fact that some horses do not lie in the stall, yet in spite of their constant standing position, they are able to rest and sleep.

The student of lameness is interested in the function of the legs in the role of supporting weight and as propelling parts, and not particularly in the capacity of these members for inflicting offense or as weapons of defense. Yet, in the exercise of their functions other than that of locomotive appliances, injury often results, but usually it is the recipient of a blow that suffers the injury, such as an animal may receive upon being kicked. Therefore, we do not often concern ourselves with strains or other injuries that the subject experiences as the result of efforts put forth in kicking or striking. Where such injuries occur, however, a diagnosis is established by making use of the principles heretofore discussed.

As propelling members the front legs bear weight and are advanced alternately when the horse is walking or trotting—in cantering this is not so. When the normal subject travels in a straight line, at a walk or a trot, the length of the stride is the same with the right and left members. The stride of the right foot then, for example, is equally divided by the imprint of the left foot, in the normal horse, when traveling at a walk and in a straight line.

Shoulder Lameness.

This enigmatical term is frequently employed by the diagnostician when he is baffled in the matter of definitely locating the cause of lameness; when he has by exclusion and otherwise arrived at a decision that lameness is "high up." Shoulder lameness may be caused by any one or several of a number of conditions, e.g., fractures of the scapula or humerus; arthritis of the shoulder or elbow joint; luxation of the shoulder or elbow joint (rarely); injuries of muscles and tendons of the region due to strains, contusions or penetrant wounds; paralysis of the brachial plexus or of the prescapular nerve; involvement of lymph glands; arterial thrombosis; metastatic infections; rheumatic disturbances; and as the result of inflammation, infectious or non-infectious occasioned by collar bruises. In some instances such inflammation is due to the manner of treatment of collar injuries. Therefore, when one considers the numerous and dissimilar possible causes of shoulder lameness, it behooves the practitioner to become proficient in diagnostic principles.

A principle which is elemental in the diagnosis of locomotory impediment, is that lameness of the shoulder or hip is usually manifested by more or less difficulty in swinging the affected member. Swinging-leg-lameness, then, is usually present in shoulder affections. In some instances lameness is mixed as in joint ailments, involvement of the bicipital bursa (bursa intertubercularis), etc. In affections of the extremity there exists supporting leg lameness. Consequently, we employ this elemental principle, and, by a visual examination of the subject, which is being made to travel suitably, one may decide that lameness is either "high up"—shoulder lameness or, "low down"—of the extremity.



To make practical use of this principle, the examiner must be thoroughly familiar with the anatomy of the various structures concerned in advancing the leg—those which support weight as well as those concerned both in weight bearing and swinging the member.

Fracture of the Scapula.

Etiology and Occurrence.—Fractures of the body of the scapula are of infrequent occurrence in horses for the reason that protection is afforded this bone because of its position. Its function, too, is such that very unusual conditions are necessary to subject it to fracture. The spine is occasionally broken due to blows such as kicks, etc., and here frequently a compound fracture exists.



Where fractures of the body of the scapula occur, heavy contusions have been the cause as a rule, and serious injury is done the subject; consequently, treatment of fracture of the body of the scapula is seldom successfully practised. Fractures of the body of this bone resulting from accidents not involving internal injury or other disturbances and which would not seriously interfere with the vitality of the subject, are not necessarily serious unless compound.

Fractures of the neck of the scapula are serious because of the fact that there occurs displacement of the broken parts and perfect apposition of the fractured ends is difficult, if not impossible.

Fractures that extend to the articular surface are very serious, and complete recovery in such instances is practically impossible. The cartilage of prolongation of the scapula is sometimes seriously involved in certain cases of fistulous withers, and in some instances it has been separated from its attachment to the rhomboidea muscles, and lameness has resulted. In such instances, the upper portion of the scapula is disjoined from all attachment, and with every movement the animal makes, the scapula is moved back and forth. Complete recovery in such cases does not occur.

Symptomatology.—Fractures of the scapular spine are ordinarily readily recognized because there is usually visible displacement of the broken part. Crepitation is also detected without difficulty.

In fractures of the body of the scapula where an examination may be made before much swelling has taken place, and in subjects that are not heavily muscled, one should have no difficulty in recognizing the crepitation.

Fractures of the neck of the scapula are recognized by crepitation, by passively moving the leg, but it is necessary to exclude fractures of the humerus when one depends upon the finding of crepitation by this means. However, unless undue swelling exists, the exact location of the crepitation is recognized without serious difficulty.

Treatment.—The treatment of compound fractures of the scapular spine consists in the removal of the broken piece of bone by way of a cutaneous incision so situated that good drainage of the wound will follow.

Simple fractures of the body of the scapula are best treated by placing the subject in a sling, if the animal is halter broken, and enforcing absolute quiet for a period of from three to six weeks. Splints or similar appliances are not of practical value in scapular fractures.

Compound fractures of the scapula usually result from violence, which at the same time does serious injury to adjacent structures, and it then becomes necessary to administer an expectant treatment, observing general surgical principles and providing in so far as possible for the comfort of the patient.

Scapulohumeral Arthritis.

Anatomy.—The scapulohumeral joint is an enarthrodial (ball and socket) joint wherein the ball or humeral articulating head greatly exceeds in size the socket or glenoid cavity of the scapula. The capsular ligament surrounding this joint is very large and admits of free and extensive movement of the articulation. There exist no lateral or common ligaments jointing the scapula and humerus as in other joints, but instead the tendinous portions of muscles perform this function. The principal ones which are attached to the scapula and humerus that act as ligaments are the supraspinatus (antea-spinatus), infraspinatus (postea-spinatus) biceps-brachii (flexor brachii) and subscapularis muscles.

Etiology and Occurrence.—Inflammation of the scapulohumeral articulation results from injuries of various kinds, including punctures which perforate the joint capsule, bruises from collars, metastatic infections and involvement as a result of direct extension of infectious conditions situated near the joint.

Classification.—Acute arthritis may be septic or aseptic, and there seems to be a remarkable tendency for recovery in cases of septic arthritis involving this joint in the horse.

Chronic arthritis with destruction of articular surfaces and ankylosis, is seldom observed. It is only in cases of severe injury, where the articular portions of the bones are damaged at the time of infliction of the injury, and where the articulation remains exposed for weeks at a time, together with immobility of the parts because of attending pain, that permanent ankylosis results.

Scapulohumeral arthritis may result then from infections, local or metastatic; from injuries, such as contusions of various kinds; from wounds, which break the surface structure or perforate the joint capsule; or from luxations.

Infectious Arthritis.

Infectious arthritis of the scapulohumeral joint the result of local causes other than produced by septic wounds, seldom causes serious inconvenience to the subject. Where such occurs, however, there is manifested mixed lameness and complete extension of the extremity is impossible. Local swelling is present and manifestations of pain are evident upon palpation of the affected area.

Treatment.—During the first stage of the infection, local applications, hot or cold, are indicated. A hot poultice of bran or other suitable material contained within a muslin sack, may be supported by means of cords or tapes which are passed over the withers and tied around the opposite fore leg. Such an appliance may be held in position more securely by attaching it to the affected member. Following the acute stage of such an infection, any local counter-irritating application or even a vesicant is in order.

Where abatement of the infectious process does not take place, and suppuration of the structures in the vicinity of the joint occurs, it is necessary to provide drainage for pus. In some cases of strangles, for instance, large pus cavities are formed and drainage is imperative. However, metastatic inflammation of this joint is seldom observed except in cases of strangles. The animal should be kept perfectly quiet until recovery has taken place.

Injuries.

Injuries to the scapulohumeral joint may be the result of kicks, runaway accidents or bruises from the collar, and there may result, because of such injuries, reactionary inflammation which will vary in intensity from the mildest synovitis to the most severe arthritis, causing more or less lameness.

Treatment.—The general plan of treatment in this form of arthritis is the same as has been outlined under the head of infectious arthritis, with the exception that there is seldom occasion to provide for drainage of pus.

Wounds.

Wounds which cause a break of the skin and fascia overlying the scapulohumeral joint are usually of little consequence, unless the blow is of sufficient force to directly injure the articulation, and in such cases, the treatment of the injury along general surgical principles, such as cleansing the area, providing drainage for wound secretion, and the administration of suitable dressing materials such as antiseptic dusting powder, is all that is required for the wound. The symptoms manifested by the subject in such cases are the same as have been discussed heretofore and merit no special consideration.

Prognosis.—Unless very serious injury be done the articular portions of the scapula or the humerus, resulting in the destruction of the capsular ligament, prognosis is entirely favorable.

Open Joint.—Where the capsular ligament is perforated and the condition becomes one of open joint, then a special wound treatment becomes necessary. The surface of the skin is first freed from all hair and filth in the vicinity of the wound. The wound proper is cleared of all foreign material either by clipping with the scissors, curetting or mopping with cotton or gauze pledgets. The whole exposed wound surface as well as the interior of the joint cavity, if much exposed, is moistened with tincture of iodin. Subsequent treatment consists in a local application of a desiccant dusting powder, which should be applied five or six times daily. The composition of the powder should be such as to permit of its liberal use, thereby affording mechanical protection to the wound as well as exerting a desiccative effect. Equal parts of boric acid and exsiccated alum serve very well in such cases.

Animals suffering from open joints of this kind should be confined in a standing position, preferably in slings, and kept so confined for three or four weeks. Since they usually bear weight upon the affected member, there is no danger of laminitis resulting.

Luxation of the Scapulohumeral Joint.

Because of the large humeral head articulating as it does with a glenoid cavity, scapulohumeral luxations are very rare in the horse. According to Moller[7], luxation is generally due to excessive flexion of the scapulohumeral joint. In such cases the head of the humerus is displaced anterior to the articular portion of the scapula and remains so fixed.

Symptoms.—Complete luxation of the scapula is recognized because of immobility of the scapulohumeral joint and of the abnormal position of the head of the humerus, which can be recognized by palpation, unless the swelling be excessive. Immobility of the scapulohumeral joint is noticeable when one attempts to passively move the parts.

Treatment.—Reduction of the luxation is effected by making use of the same general principles that are employed in the reduction of all luxations, and they are—the control of the animal so that the manipulations of the operator are not antagonized by muscular contraction, which is best accomplished by anesthesia; placing the luxated bones in the position which they have taken to become unjointed; and then making use of force which is directed in a manner opposite to that which has effected the luxation.

In a forward luxation of this kind, the operator should further flex the humerus, and while it is in this flexed position, force is exerted upon the articular head of this bone, and it is pushed downward and backward into its normal position.

After-care consists in restriction of exercise and, if necessary, confining the subject in a sling and the application of a vesicant over the scapulohumeral region.

Inflammation of the Bicipital Bursa. (Bursitis Intertubercularis.)

Anatomy.—There is interposed between the tendon of the biceps brachii (flexor brachii) and the intertubercular or bicipital groove a heavy cartilaginous pad, which is a part of the bursa of the biceps brachii. This synovial bursa forms a smooth groove through which the biceps brachii glides in the anterior scapulohumeral region. Great strain is put upon these parts because the biceps brachii is the chief inhibiting structure of the scapulohumeral articulation—the one which prevents further flexion of the humerus during weight bearing. Passing, as it does, over two articulations, the biceps brachii has a somewhat complicated function, being a flexor of the radius and an extensor of the humerus. Thus it is seen, the biceps brachii is a weight bearing structure, as well as one that has to do with swinging the leg.

Etiology and Occurrence.—Because of the exposed position of the bicipital bursa (bursa-intertubercularis) it is occasionally injured. Blows and injuries received in runaway accidents do serious injury to the bursa and because of the peculiar and important part it plays during locomotion, serious injuries are not likely to resolve, and too often chronic lameness results. It is to be noted that the tendon of the biceps brachii (flexor brachii) is always involved in cases of inflammation of the bicipital bursa, and according to the late Dr. Bell[8] strain of the biceps brachii is a frequent cause of lameness in city horses, more frequent than is generally supposed.

Pathological Anatomy.—More or less destruction of the cartilaginous portion of the bursa, sometimes involving the tendinous portion of the biceps, takes place and, according to Moller, in some instances there occurs ossification of the tendon. Autopsies in some old horses reveal the presence of erosions of cartilage and hyperthrophy of the inflamed parts.

Symptoms.—In acute inflammations, there is always marked lameness. This is manifested to a greater degree when the subject advances the affected leg. There is incomplete advancement of the member; the toe is dragged when the horse is made to walk and the foot kept in a position posterior to the opposite or weight bearing foot while the subject is at rest. Lameness is disproportionate to the amount of local manifestation in the way of heat, swelling and pain that is to be recognized on palpation. In fact, in some cases so much pain attends the condition that no weight is borne by the affected member, and when compelled to walk, the subject hops on the sound leg.

Chronic inflammation of the bicipital bursa is occasionally met with wherein both members are affected. Because of the nature of the structures involved, when inflamed, chronic inflammation is a more frequent termination than is complete recovery. Bilateral affections are seen in horses that are driven for years, regularly at a fast pace on paved streets. In such cases, the gait is stilted, that is, there is incomplete advancement of both members and, of course, the period of weight bearing is correspondingly shortened; hence the short strides.

In chronic cases, little if any evidence of inflammation is to be detected by digital manipulation of the parts. If flinching occurs, one is often unable to interpret the manifestation as to whether it is due to inflammation or not.

There is no marked "warming out" in this condition, and animals are nearly as lame after having been driven a considerable distance as when started, although the lameness is not as a rule very great.

Treatment.—In very painful cases acute inflammation is treated by employing cold applications during the initial stage. Cracked ice when contained in a suitable sack may be held in contact with the affected part and the pack is supported by means of cords or tapes as suggested in the discussion on treatment of scapulohumeral arthritis on page 66. Later, hot applications may be employed to good advantage.

In the course of ten days or two weeks, if the acute painful condition has entirely subsided, vesication is indicated. The ordinary mercury and cantharides combination does very well. Depending upon the course taken in any given case, one is guided in the treatment employed. If prompt resolution comes to pass, the subject may be given free run at pasture after three or four weeks confinement in a box stall. If, however, the case does not progress in a prompt and satisfactory manner, absolute quiet must be enforced for six weeks or more. Repeated blistering is beneficial, although it is doubtful if firing is of sufficient benefit in the average chronic case of intertubercular bursitis to justify the punishment which this form of treatment inflicts, unless infliction of pain is the thing sought, to enforce repose in restless subjects. Patients are best given a long rest at pasture and returned to work for two or three months after an acute attack of inflammation of the bursa, lest the condition become chronic. When due consideration is given the pathology of such cases, the frequent unsatisfactory termination under the most careful treatment, is readily understood.

Contusions of the Triceps Brachii. (Triceps Extensor Brachii: Caput Muscles.)

Anatomy.—The triceps brachii is the principal structure which fills the space between the posterior border of the scapula and the humerus. The several heads originate for the most part on the border of the scapula, the deltoid tuberosity of the humerus and the shaft of the humerus. Insertion of this large muscular mass is effected by means of several tendons to the olecranon. A synovial bursa is situated underneath the tendinous attachment of the posterior portion of the triceps brachii—the long head or caput magnum.

The function of the triceps as a whole is to flex the shoulder joint and extend the forearm. The triceps brachii is the chief antagonist of the biceps brachii.

Etiology and Occurrence.—Owing to the exposed position of this structure, it is not infrequently contused, the result of falls, kicks and other injuries. The function of the triceps is such that it becomes strained upon rare occasions when a horse resists confinement of restraint in such manner that the parts are unduly tensed in contraction. This sort of resistance may stretch the radial nerve or its branches in a way that paralysis results. A condition known as "dropped elbow" is described by Henry Taylor, F.R.C.V.S., in the Veterinary Record[9], wherein a two-year-old colt while resisting confinement was so injured.

The triceps group because of its convenient location, constitutes the site for hypodermic injection of drugs and biologic agents, with some practitioners; and as a result, more or less inflammation may occur. The author has observed and treated some twenty cases where an intensely painful infectious inflammation of the triceps brachii was caused by the intramuscular injection of a caustic solution by a cruel and unscrupulous empiric, whose object was to increase his practice.

Symptomatology.—As the triceps brachii is not particularly taxed during weight bearing in the subject at rest, there may be no unnatural position assumed during inflammation of the triceps. More or less swelling and supersensitiveness is always present, however, and great care and discrimination must be exercised in digital manipulation of the triceps region because many animals are normally sensitive to palpation of these parts. It is sometimes difficult to correctly interpret the true state of conditions because of this peculiarity.

There is always swinging-leg-lameness, which is accentuated when the subject is urged to trot. Where symptoms are pronounced, it is unnecessary to cause the subject to move at a faster pace than at a walk to recognize the condition. The forward stride is shortened and in extremley painful conditions, no attempt is made to extend the leg. It is simply carried en une piece—flexion of the shoulder and elbow joints is carefully avoided.

Treatment.—During the early stage of inflammation, hot or cold applications are beneficial. Long continued use of moist heat—fomentations—allays pain and stimulates resolution. Keeping in contact with the painfully swollen parts a suitable bag filled with bran, which can be moistened at intervals with warm water, constitutes a practical and easy means of treatment. By employing this method, one is more likely to succeed in having his patient properly cared for, in that less work is entailed than if hot fomentations are prescribed.

After the acute and painful stage has subsided, a stimulating liniment is of benefit. The subject should be kept within a comfortable and roomy box stall for a sufficient length of time to favor prompt resolution. Wild and nervous subjects, if not so confined, will probably overexert the affected parts if allowed the freedom of a paddock or pasture.

Where the inflammation becomes infective, surgical interference is necessary. The prompt evacuation of pus, with adequate provision for wound discharge, should be attended to before extensive destruction of tissue takes place. Resolution is prompt as a rule in such cases because of the vascularity of the structures and the ease with which proper drainage may be effected. No special after-care is necessary if drainage is perfect, except that one should avoid injecting the wound cavity with aqueous solutions unless it be absolutely necessary to cleanse such cavity, and then it is best to swab the wound rather than to irrigate it freely.

Shoulder Atrophy. (Sweeny or Swinney)

No satisfactory consideration of the pathogeny of this condition is recorded, but practitioners have long distinguished between muscular atrophies which are apparently caused without doing serious injury to nerves and muscular atrophy which seems to be due to nerve affection. In the first instance, recovery when proper attention is given, is prompt; whereas, in the latter, regeneration of the wasted tissues requires months in spite of the best sort of treatment.

The parts more frequently affected are the supra- and infrascapularis (antea- and posteaspinatus) muscles. But in some cases the triceps group is involved; however, this occurs in unusual and chronic affections. No doubt, these chronic cases are due to suspended innervation and are not to be classed with the ordinary case of atrophy of the abductor muscles of the humerus (supra- and infraspinatus) as in the usual case of "sweeny."

Occurrence.—Shoulder atrophy such as the general practitioner commonly meets with, is an affection, more often seen in young animals and it seems to be due to injuries of various kinds which contuse the muscles of the shoulder. Ill-fitting collars and pulling in a manner that there occurs side draft with unusual strain on the muscles of one side of the neck and shoulder, seem to be the more frequent causes of this trouble. Blows such as are occasioned by kicks and falls frequently result in atrophy of shoulder muscles.

Course.—In some cases a rapidly progressive atrophy characterizes the case and lameness and atrophy appear at about the same time. The affection in such instances does not recover spontaneously but constitutes a condition which requires prompt and rational treatment so that function may be fully restored to the parts involved.

Occasionally one may observe cases where there is but slight atrophy; where the disease progresses slowly and atrophy is not extensive or marked. In vigorous young animals that are left to run at pasture when so mildly affected, spontaneous recovery occurs.

Symptomatology.—Lameness is the first manifestation of shoulder atrophy, and in many cases where lameness is slight, the veterinarian may fail to discover the exact nature of the trouble if he is not very proficient as a diagnostician of lameness or if he is careless in taking into consideration obtainable history, age of the subject, etc. Because of the fact that the average layman believes that practically every case of fore-leg lameness wherein it is not obvious that the cause is elsewhere, is due to a shoulder affection of some kind, we may be too hasty in giving the client assurance that no "sweeny" exists. In some of these cases where a diagnosis of "shoulder lameness" has been made and the client has been assured that no sweeny exists, the patient is returned in about a week and there is then marked atrophy of one or both of the spinatus muscles.

A mixed type of lameness characterizes this affection, and in the average case there exists little evidence of local pain. The salient points in recognizing the condition are a consideration of history if obtainable; age of the subject; finding slight local soreness, by carefully manipulating the muscles which are usually involved; noting the character of the lameness if any is present; and where atrophy is evident, of course, the true condition is obvious.

Treatment.—Subcutaneous injections of equal parts of refined oil of turpentine and alcohol, with a suitable hypodermic syringe, is a practical and ordinarily effective treatment. From five to fifteen cubic centimeters (the quantity varies with the size of the animal), of this mixture is injected into the atrophied parts at different points, taking care to introduce only about one to two cubic centimeters at each point of injection. The syringe should be sterile and, needless to say, the site of injections must be surgically clean.

Other agents, such as tincture of iodin, solutions of silver nitrate, saline solutions and various more or less irritating preparations have been employed; but in the use of these preparations one may either fail to stimulate sufficient inflammation to cause regeneration to take place, or infection is apt to occur. Where suppuration results, surgical evacuation of pus must be promptly effected else large suppurating cavities form.

The employment of setons constitutes a dependable method of treatment of shoulder atrophy, but because of the attendant suppurative process which inevitably results, this method is not popular with modern surgeons and is a last resort procedure.

After-care.—Regular exercise such as the horse usually takes when at pasture, is very helpful in treating atrophy, and in some cases it has been found that no reasonable amount of irritation would stimulate muscular regeneration; but by later allowing patients to exercise at will, recovery took place in a satisfactory manner. No special attention is ordinarily necessary.

Paralysis of the Suprascapular Nerve.

Anatomy.—The suprascapular (anterior scapular) nerve, a small branch of the brachial plexus, is given off from the anterior portion of this plexus. The nerve rounds the anterior border of the neck of the scapula, passing upward and backward under the supraspinatus (antea-spinatus) muscle and terminating in the infraspinatus (postea-spinatus) muscle.

Etiology and Occurrence.—As the result of direct injury to this nerve by contusion such as may be received in runaway accidents, collar bruises, especially collar bruises in young horses that are not accustomed to pulling and that walk in a manner to cause side draft, injury to the nerve occurs, and partial or complete paralysis supervenes. Some writers state that it may be produced by confining an animal in recumbency, with the casting harness. The common cause of paralysis or paresis of this nerve in cases such as one observes in country practice, is bruises from the collar in colts that are put to heavy farm work or where ill fitting collars are used.

Symptomatology.—With partial or complete suspension of function of the suprascapular nerve there results enervation of the supraspinatus and infraspinatus muscles. Since these muscles act as external lateral ligaments of the scapulohumeral joint, when they are incapacitated, there naturally follows more or less abduction of the shoulder when weight is borne.

In extreme cases, as soon as the ailing animal is caused to support weight with the affected member, the joint is suddenly thrown outward in a manner that the average layman at once concludes that there must be scapulohumeral luxation, and the veterinarian receives a call to see a case wherein the "shoulder is out of place." There exists, however, no luxation in such cases.

If serious injury is done the nerve so that it undergoes degenerative changes, there will result atrophy of the muscles that derive their nerve supply from the suprascapular nerve.



Treatment.—During the first few days following injuries which result in this form of paralysis, it is well to keep the subject inactive, and if much inflammation of the injured structures contiguous to the nerve exists, the application of cold packs is beneficial. Later, as soon as acute inflammation has subsided, vesication of a liberal area around the anteroexternal part of the scapulohumeral joint and over the course of the suprascapular nerve, will stimulate recovery in favorable cases. As a rule, in mild cases, the subject is in a condition to return to work in two or three weeks.

Radial Paralysis.

Described under the titles of "Radial Paralysis" and "Brachial Paralysis," there is to be found in veterinary literature a discussion of conditions which vary in character from the almost insignificant form of paresis to the incurably affected conditions wherein the whole shoulder is completely paralyzed.

When one considers the anatomy of the brachial nerve plexus and the distribution of its various branches, the location of this plexus and its proximity to the first rib, and the inevitable injury it must suffer in fracture of this bone, together with the inaccessibility of the plexus, it is not strange that a correct diagnosis of the various affections of the brachial plexus and the radial nerve is often impossible until several days or weeks have passed. And, in some instances, diagnosis is not established until an autopsy has been performed. Here, too, we fail to find cause for paralysis in some rare instances.

Anatomy.—The radial nerve is a large branch of the brachial plexus and is chiefly derived from the first thoracic root of the plexus and is here situated posterior to the deep brachial artery. It is directed downward and backward under the subscapularis and teres major muscles, rounding the posterior part of the humerus, and passing to the anterior and distal end of the humerus, it finally terminates in the anterior carpal region. The radial nerve supplies branches to the three heads of the triceps brachii, to the common and lateral extensors of the digit and also to the skin covering the forearm.

Etiology and Occurrence.—Nothing definite is known about the cause of some forms of radial paralysis. However, radial paralysis is encountered following injury to the nerve occasioned by its being stretched, as in cases where the triceps brachii is unduly extended in restraining subjects by means of a casting harness. Berns[10] states that in confining horses on an old operating table where it was necessary to draw the affected foot forward twenty-four to thirty-six inches in advance of its fellow, which was secured in a natural vertical position, radial paralysis of a mild form was of frequent occurrence. Country practitioners, in restraining colts by casting with harness or ropes, occasionally observe a form of paresis wherein the radial nerve suffers sufficient injury that there is caused a temporary loss of function of the triceps brachii. Such cases recover within three or four days and are not a true paralysis, but nevertheless constitute conditions wherein normal nerve function is temporarily suspended.



Symptoms.—Immediately subsequent to injuries which involve the radial nerve, there is manifested more or less impairment of function. Remembering the structures supplied by the radial nerve and its branches, one can readily understand that there should occur as Cadiot[11] has stated:

In complete paralysis, the joints of the affected limb with the exception of the shoulder are usually flexed when the horse is resting. In consequence of loss of power in the triceps and anterior brachial muscles, the arm is extended and straightened on the shoulder, the scapulohumeral angle is open, and the elbow depressed. The forearm is flexed on the arm by the contraction of the coracoradialis (biceps brachii), while the metacarpus and phalanges are bent by the action of the posterior antibrachial muscles. The knee is carried in advance, level with, or in front of, a vertical line dropped from the point of the shoulder. The hoof is usually rested on the toe, but when advanced beyond the above mentioned vertical line, it may be placed flat on the ground, the joints then being less markedly bent. When the limb as a whole is flexed, it may be brought into normal position by thrusting back the knee with sufficient force to counteract the action of the flexor muscles.



When made to walk, the animal being unable to exert muscular action with the paralyzed structures, limply carries the member as a whole, and there is shortening of the anterior portion of the stride. There being loss of function of the triceps brachii, it is impossible for the subject to straighten the leg in the normal position for supporting weight; therefore, any attempt to bear weight results in further flexion of the affected member and the animal will fall if the body is not suddenly caught up with the sound leg.

Differential Diagnosis.—In making examination of these cases, one can exclude fracture by absence of crepitation and usually, also, swelling is absent in radial paralysis. In a typical case of radial paralysis, the affected leg can sustain its normal share of weight if placed in position, that is, if the carpal joint is extended in such manner that the leg is positioned as in its normal weight-bearing attitude. In brachial paralysis, whether due to fracture of the first rib or to other serious injury, it is impossible for the subject to support weight with the affected member even when it is passively placed in position.

No difficulty is ordinarily experienced in differentiating radial paralysis from muscular injuries to the triceps; yet, in some cases of "dropped elbow," it is necessary to observe the progress of the case for ten days or two weeks before one can positively establish a diagnosis.

Quoting Merillat[12]: "When, after four weeks, there is no amelioration of the paralysis, the muscles have atrophied, and the patient has become emaciated from pain and discomfort, the diagnosis of brachial paralysis with fracture of the first rib may then be announced."

Prognosis.—When no complete paralysis of the brachial plexus or no fracture of the first rib exists, the majority of cases recover completely in from ten days to six weeks. Some writers claim that recoveries occur in ninety per cent of cases when conditions are favorable.

Treatment.—When incomplete radial paralysis exists, little needs be done except to allow the subject moderate exercise and to provide for its comfort. Local applications, stimulative in character, are beneficial, and the internal administration of strychnin is indicated.

In the cases where weight is not supported without the affected leg being passively placed in position, it is necessary to provide for the subject's comfort in several ways.

Mechanical appliances such as braces of some kind in order to keep the affected leg in a position of carpal extension, constitute the essential part of treatment. The leg is supported in such a manner that flexion of the carpus is impossible. Due regard is given to prevent chafing or pressure necrosis by contact of the skin with the braces—this may be done by bandaging with cotton. The supportive appliance is kept in position for ten days or two weeks. At the end of this time the brace may be removed and the subject given a chance to walk, and improvement, if any exists, will be evident. When there is manifested an amelioration of the condition, moderate daily exercise and massage of the affected parts are helpful.

Should the subject be seriously inconvenienced by the application of a brace or other supportive appliances, it is necessary to employ slings. Further, if weight is supported entirely by the unaffected member, laminitis may supervene if a sling is not used.

Thrombosis of the Brachial Artery.

Thrombosis of the brachial artery or of its principal branches is of very rare occurrence in horses.

Etiology.—Partial or complete obstruction of arteries (brachial or others) occurs as the result of direct injury to the vessel wall from compression and tension of muscles and resultant arteritis; lodging of emboli; and parasitic invasion of vessel walls causing internal arteritis.

Symptomatology.—If sufficient collateral circulation exists to supply the parts with blood, no inconvenience is manifested while the subject is at rest. Where the lumen of the affected vessel is not completely occluded, there may be no manifestation of lameness when the ailing animal is moderately exercised. Consequently, the degree of lameness depends upon the extent of the obstruction to circulation; and, likewise, the course and prognosis depend upon the character and extent of such obstruction.

In severe cases, lameness is markedly increased by causing the animal to travel at a fast pace for only a short distance. There are evinced symptoms of pain, muscular tremors and sudation, but the affected member remains dry and there is a marked difference of temperature between the normal areas and the cool anemic parts. When the subject is allowed to rest, circulation is not taxed, and there is a return to the original and apparently normal condition, only to recur again with exertion. This condition characterizes thrombosis.

Treatment.—In these cases, little if any good directly results from any sort of treatment in the way of medication. Absolute rest is thought to be helpful. Potassium iodid, alkaline agents such as ammonium carbonate and potassium carbonate, have been administered. Circulatory stimulants also have been given, but it is doubtful if any good has come from medication.

Fracture of Humerus.

The shaft of the humerus, protected as it is by heavy muscles, is not frequently fractured; and fractures of its less protected parts, as for example, the head, are complicated in such manner that resultant arthritis soon constitutes the more serious condition.

As a result of falls on frozen ground, kicks or any other form of heavy contusion, the humerus is occasionally broken. It is rarely fractured otherwise. Because of the force of contusions usually required to effect humeral fracture, the manner in which the bone is broken, with respect to direction, is variable. Often oblique fractures exist and occasionally there occurs multiple fracture. In addition to the ordinarily serious nature of the fracture itself, there is always much injury done the adjoining structures.

Symptomatology.—Mixed lameness and manifestation of severe pain characterize this affection. Considerable swelling which increases, in some cases for a week or more, is to be observed. Crepitation is readily detected, if pain and swelling is not too great to prevent passive movement of the member. Where intense pain is not manifested, because of manipulation, one may abduct the extremity and thereby occasion distinct crepitation; but when it is possible to recognize crepitation by holding the hand in contact with the olecranon while the animal is made to walk, this method is to be preferred, if the subject can move without serious difficulty. The pathognomonic symptom here is recognition of crepitation, but this may be very difficult to recognize in fracture of condyles, and in such instances, a careful examination is necessary. Gentle manipulation in a manner that pain is not aggravated will tend to inspire confidence on the part of the subject and relaxation of muscles will enable the operator to detect crepitation.

Course and Prognosis.—Because of the direction of the long axis of the humerus, with relation to the bony column of the extremity, it is obvious that any lateral movement of the leg tends to rotate the shaft of this bone. In fractures of the shaft of the humerus, then, it is apparent that immobilization is very difficult if at all possible.

The proximity to the axillary lymph glands makes for easy dissemination of infection when the contused musculature becomes infected. The adjacent brachial nerve plexus is so very apt to become involved, if not actually injured at the time fracture occurs, that paralysis is a probable complication. Consequently, it is logical to reason that because of the many possible serious complications, such as shock, occasioned by the injury and the distress and pain which this accident produces, recovery must be the exception in fracture of the humerus. However, recoveries do take place and in addition to the reported recoveries by Liautard, Moller, Stockfleth, Lafosse, Frohner and others, we have instances cited by American practitioners where cases resulted in recovery. Thompson[13] reports a good recovery in a 1600-pound mare where there existed an oblique fracture of the humerus. This mare was kept in slings for eight weeks. Walters[14] reports complete recovery in humeral fracture in a foal three days old. The only treatment given was the application of a pitch plaster from the top of the scapula to the radius. The colt was kept in a comfortable box stall and in about four weeks regained use of the leg. Complete recovery eventually resulted. In the experience of the author, recovery has not occurred in humeral fractures.

Treatment.—When animals are not aged and of sufficient value to justify treatment, they are best supported in a sling, if halter broken. If subjects are nervous, wild and unbroken, it is possible to employ the sling, if care is given to train the animal to this manner of restraint. The presence of an attendant for a day or two will reassure such subjects so that even in these cases it may be practicable to employ the sling.

Braces and other mechanical appliances intended to immobilize the parts are not of practical benefit in the horse. Unlike the dog, the horse as yet has not been successfully subjected to tolerating rigid braces for the shoulder and hip.

Everything possible must be done that will make for the patient's comfort. If the subject turns out to be a good self nurse, and the nature of the fracture is such that practical apposition of the broken ends of bone may be maintained, recovery will occur in some cases.

Inflammation of the Elbow. (Arthritis.)

Affections of this articulation other than those which are produced by traumatism are rare. This joint has wide articular surfaces, and securely joined as they are by the heavy medial and lateral ligaments (internal and external lateral ligaments), luxation is practically impossible. When luxation does occur, irreparable injury is usually done. Castagne as quoted by Liautard[15], reports a case of true luxation of the elbow joint in a horse where reduction was effected and complete recovery took place at the end of twenty-five days. This is an unusual case. The average practitioner does not meet with such instances.

Anatomy.—The condyles of the humerus articulate with the glenoid cavities of the radius and a portion of the ulna. Two strong collateral ligaments pass from the distal end of the humerus to the head of the radius. The capsular ligament is a large, loose membrane which encloses the articular portion of the humerus with the radius and ulna and also the radioulnar articulation. It is attached anteriorly to the tendon of the biceps brachii (flexor brachii). The capsule extends downward beneath the origin of these digital flexors. This fact should be remembered in dealing with puncture wounds in the region, lest an error be made in estimating their extent and an open joint be overlooked at the initial examination.

Etiology and Occurrence.—Exclusive of specific or metastatic arthritis, which is seldom observed except in young animals, inflammation of the elbow joint is usually caused by injury. This articulation is not subject to pathologic changes due to concussion or sprains as occasioned by ordinary service, but is frequently injured by contusion from falls, blows from the wagon-pole and kicks. Wounds which affect the elbow joint, then, may be thought of in most cases, as resultant from external violence. They may be contused wounds or penetrant wounds. Sharp shoe-calks afford a means of infliction of penetrant wounds which may occasion open joint and infectious arthritis.

Classification.—A practical manner of classifying inflammation of the elbow is on an etiological basis. Eliminating the forms of elbow inflammation, such as are caused by metastatic infection and other conditions which properly belong to the domain of theory of practice, we may consider this affection under the classification of contusive wounds and penetrative wounds.

Symptomatology.—Any injury which is of sufficient violence to occasion inflammation of the elbow causes marked lameness and manifestation of pain. The degree of lameness and distress manifested by the subject, depends upon the nature and extent of the involvement. A contusion suffered as the result of a fall, which occasions a circumscribed inflammation of the structures covering this joint and where little inflammation of the articulating parts exists, marked evidence of pain and lameness might be absent. On the other hand, if a true arthritis is incited, there will be evident distress manifested, such as hurried respiration, accelerated pulse, inappetence, mixed lameness, local evidence of inflammation and particularly marked supersensitiveness of the affected parts. Considering these two extremes of manifested distress and injury, one may readily conclude that in the frequently seen case, wherein contusion has occasioned a moderate degree of injury, prognosis is favorable and recovery ordinarily follows in the course of a few weeks' treatment.

In cases of arthritis due to penetrative wounds (because of the important function of this joint and its large capsule, which when inflamed discharges synovia in a manner that closure of such an open joint is seldom possible) a very grave condition results.

Treatment.—Inflammation of the elbow, such as is frequently seen in general practice where horses are turned out together and exposed to kicks and other injuries, yields to treatment readily, if an open joint does not exist.

Hot packs supported in contact with the elbow and kept around the inflamed articulation for a few days, materially decrease pain and tend to reduce inflammation. The subject must be kept quiet in a comfortable stall and, if necessary, a sling used. Where it is impossible for the animal to support much weight with the injured member the sling should be employed.

As inflammation abates, which it does in the course of from one to three weeks in uncomplicated cases, the subject may be allowed the freedom of a comfortable box stall. Vesication of the parts is in order, and this may be repeated in the course of two weeks, if it is deemed necessary.

Penetrative wounds resulting in open joint are not treated with success as a rule, and because of the handicap under which veterinarians labor, methods of handling such cases, where large, important articulations are affected, are not being rapidly improved. Prognosis is usually unfavorable, and for humane and economic reasons, animals so affected should be destroyed.

Ordinary wounds of the region of the elbow are treated along general lines usually employed. They merit no special consideration, except that it may be mentioned that with such injuries concomitant contusion of the parts occasions injury that does not recover quickly.

Fracture of the Ulna.

Etiology and Occurrence.—Fractures of the ulna in the horse are not common in spite of the exposed position of the olecranon. This bone when broken, is usually fractured by heavy blows and any form of ulnar fracture is serious because of its function and position in relation to the joint capsule. Transverse fractures do not readily unite because of the tension of the triceps muscles, which prevent close approximation of the broken ends of the bone.

Thompson[16], however, reports a case of transverse simple fracture of the ulna in a mare, the result of a kick, in which complete recovery took place. He kept the subject in a sling for six weeks and then allowed six months rest.

Symptomatology.—The position assumed by a horse suffering from a transverse fracture of the ulna, is similar to that in radial paralysis. Crepitation may be detected by manipulating the parts, and in some instances of fracture of the olecranon, there occurs marked displacement of the broken portions of the bone. Lameness is intense and the parts are swollen and supersensitive. The capsular ligament of the elbow joint is usually involved in the injury because fracture of the ulna may directly extend within the capsular ligament. In such cases, there is synovitis, and later arthritis causes a fatal termination.

Treatment.—The impossibility of applying a bandage in any way to practically immobilize these parts in fracture of the ulna, prevents our employing bandages and splints. Therefore, one can do little else than to put the patient in a sling and try to keep it quiet and as nearly comfortable as circumstances allow.

Fracture of the Radius.

Etiology and Occurrence.—From heavy blows received such as kicks, collision with trees or in falls in runaway accidents, the radius is occasionally fractured. In very young foals, fracture of the radius, as well as of the tibia and other bones, results from their being trampled upon by the mother.

Symptomatology.—Excepting in some cases of radial fracture of foals where considerable swelling has taken place, there is no difficulty in readily recognizing this condition. The heavy brachial fascia materially contributes to the support of the radius, and in cases where swelling is marked, crepitation may not be readily detected. In fact, a sub-periosteal fracture may exist for several days or a week or more and then, with subsequent fracture of the periosteum, crepitation and abnormal mobility of the member are to be recognized. In such cases, the subject will bear some weight upon the affected member, but this causes much distress. In one instance the author observed a transverse fracture of the lower third of the radius which was not positively diagnosed until about ten days after injury was inflicted. In this case, without doubt, the subject originally suffered a sub-periosteal fracture of the bone and because the animal was a good self nurse, the brachial fascia supported the radius until the periosteum gave way and the leg dangled. In this instance infection took place and suppuration resulted. It was deemed advisable to destroy this animal.

Prognosis.—In adult animals, radial fracture constitutes a grave condition; generally speaking, prognosis, in such cases, is unfavorable. Because of the leverage afforded by the extremity, immobilization of the radius is difficult. Any sort of mechanical appliance, which will immobilize these parts, is likely to produce pressure-necrosis of the soft structures so contacted. There is occasioned thereby much pain and the subject becomes restive, unmanageable and sometimes the splints are completely deranged because of the animal's struggles, and much additional injury to the leg is done. Occasionally, an otherwise favorable case is thus rendered hopelessly impossible to handle, and the subject must be destroyed several days after treatment has been instituted.

Consequently, unless all conditions are good, and the affected animal a favorable subject, young, of good disposition, and the fracture a simple transverse one, complete recovery is not likely to result from any practical means of handling.

Treatment.—Mature subjects ought to be put in slings and kept so restrained throughout the entire time of treatment. Immobilization of the broken parts of the bone is the object sought. This is attempted by practitioners who employ various methods, and each method has its advocates.

Casts are used by some and serve very well in many cases; but because of their bulk and unyielding and rigid nature, they are not well adapted to use on fractures of bones proximal to the carpus and tarsus. This is in reference to plaster-of-paris casts or those of any similar material.

Appliances which depend on glue or other adhesive substances combined with leather, wood or fiber for their support, are efficacious but not comfortable.

The use of heavy leather when the member has been suitably padded with cotton and bandages, constitutes a very good manner of reducing fracture of the radius or of the tibia. Leather when cut to fit both the medial and lateral sides of a leg, and firmly held with bandages, will form a firm support that yields slightly to changes of position, thus making for comfort of the subject.

Such a splint or support should extend from the fetlock region to the elbow, but the cotton and bandages are to reach to the foot. When one considers that, with the supportive appliance placed on each side of the affected member, rigidity is accomplished as much from tensile strain put upon the leather as from its own stiffness, it is seen that the leather need not be of the heaviest—sole leather is unnecessary. Because of the more comfortable immobilizing appliance, the subject is less restive, and chances for a successful outcome are materially increased thereby.

In the mature subject, six or eight weeks' time is required for union of the parts to occur sufficiently so that splints may be dispensed with. Rearrangement of the supportive apparatus, however, is possible and usually necessary during the first few weeks of treatment. By employing care in handling the parts, the subject will be unlikely to do itself injury at the time readjustment of splints is being effected.

In foals, it is best to give them the run of a box stall with the mother. Being agile, they get up and lie at will without doing injury to the fractured member. The splints (leather is preferable in these cases also) are looked after and readjusted as necessity demands.

Three or four weeks time is all that is required for the average young colt to be kept in splints when suffering from simple transverse fracture of the radius.

Compound fractures are necessarily more difficult to treat than are the simple variety, but even in such cases recovery results sometimes, and the practitioner is justified in attempting treatment after having explained the situation to his client.

Oblique fractures, even when simple, do not completely recover. Muscular and tendinous contraction, together with the natural tendency for the beveled contacting parts of the broken bone to pass one another in oblique fracture, results in shortening of the leg and, if union results, a large callus usually forms. Where shortening of bones occur, necessarily, permanent lameness follows.

Wounds of the Anterior Brachial Region.

Etiology and Occurrence.—Contusions and lacerations of the forearm are of frequent occurrence in horses and are troublesome cases to handle; particularly is this noticeable where extensive laceration of the parts occurs. These injuries are caused by animals being kicked; by striking the forearm against bars in jumping; and in sections of the country where barbed wire is used to enclose pastures, extensive lacerated wounds are met with when horses jump into such fences.

Symptomatology.—Any wound which causes inflammation of the structures of the anterior half of the forearm, is characterized by swinging-leg-lameness. Depending upon the nature and extent of the injury, manifestation varies. In cases where laceration has practically divided all of the substance of the extensor tendons, it is, of course, impossible for the subject to advance the leg; but where lacerated wounds involve only a part of the extensor apparatus of the foreleg, not so much inconvenience is evident, unless the wound is seriously infected and inflammation involves contiguous structures. Therefore, in many instances, lameness is more pronounced in contusions of the anterior brachial region than where tissues have been divided more or less keenly.

In every instance diagnosis is easily established. The injury is quite evident, and the manner of locomotion is not in itself an essential feature to be considered in a discussion of symptoms. Where a contusion of the anterior brachial structures occurs, there is, in addition to lameness, swelling which is painful because of the pressure occasioned by the heavy non-yielding brachial fascia. And where suppuration occurs, there is then an intensely painful condition which is not relieved until pus has been evacuated. Rather frequently, drainage for wound secretions is a difficult problem, and approximation of the divided ends of muscles is always difficult to maintain.

Treatment.—Contused wounds of the anterior brachial region are treated along usual lines; that is, attempt is made to stimulate prompt resolution. Hot or cold applications are employed throughout the acute stage of the affection. Complete rest is provided for until all pain has subsided. Later, stimulating liniments are beneficial.

Where no injury is done the periosteum or bone, complete resorption of all products of inflammation usually occurs, though in many instances, this is tardy—six weeks or more are sometimes required for recovery to take place.

If suppuration occurs, it is necessary to provide for drainage as soon as it is possible to distinguish the presence of pus. Due regard is given the manner of establishing drainage because of the usual existence of sub-fascial fistulae. In these cases, one avoids injecting solutions of aqueous antiseptics. By gently compressing the parts, pus is caused to drain out and in enforcing a moderate amount of exercise at a walk, when lameness is not intense, drainage is maintained. Cotton packs, moistened with hot antiseptic solutions, and kept around the forearm for several hours daily, are helpful because drainage is facilitated, and resolution is stimulated by the increase of blood thus attracted to the parts, and pain materially diminishes.

In lacerated wounds of the anterior brachial region, after having controlled hemorrhage, an area around the wound margin is freed of hair by clipping or shaving. The wound is carefully examined, and the best site for drainage is selected and a suitable opening for wound discharge is provided for. Where the extensor carpiradialis (metacarpi magnus) with other structures, is divided and the distal portion is torn downward, as frequently is the case in barbed wire cuts, it is necessary to make careful provision for drainage. The wound is thoroughly cleansed by means of ablutions if necessary; but preferably by swabbing with pledgets of cotton or gauze which are moistened in antiseptic solutions. All shreds of macerated tissue are clipped with scissors and finally the whole wound surface is painted with tincture of iodin.

If drainage is made by cutting through the tissues in the median portion of the structures that have been displaced, the opening should be packed with gauze so that it may remain patent after swelling has occurred. Such packing is left in situ for twenty-four hours.

The pendant muscular portions of tissues are sutured up by means of tapes and, while perfect apposition is not ordinarily possible, it is very essential to train the pendant tissues in their normal position even if they require resuturing within a week. This minimizes granulation of tissue, and there results less scar if the detached portions are kept near, even if not in contact with the proximal wound margins. The skin together with subcutaneous fascia is sutured on either side unless drainage is to be provided for on one side, and the lowermost part of that side is left unsutured.

After-care.—Where extensive suturing of tissues has been necessary, subjects must be kept quiet. They are best confined in box stalls and not taken out for several weeks. Particularly is this true where transverse division of extensors has taken place. Sutures are removed at the end of from ten days to three weeks as cases permit. Drainage of wound secretions, which usually become infected, is necessary, because with obstructed drainage in an infected wound of this kind, there will result an early destruction of tissue at some point sutured. Daily irrigation done in a manner that practical asepsis is carried out, is necessary for about a week. All irrigation is done by way of the drainage opening, and this with warm aqueous solutions of suitable antiseptics. After a week or ten days' time, the wound should not be dressed more frequently than twice weekly.

If it is necessary to leave a portion of the wound uncovered, as in cases where skin is destroyed, the frequent (three or four daily) application of a suitable antiseptic powder is necessary to check exuberant granulation. This may be directly effected by the use of an astringent or desiccant preparation, and such dressing serves as a mechanical protection as well.

When such wounds are kept clean, where drainage is properly maintained, and the subject kept quiet, no particular attention other than the local application of an astringent lotion (such as the zinc and lead lotion) is necessary after the first three or four weeks. Usually, if the animal gnaws at the parts or otherwise manifests evidence of discomfort, it is an indication that new areas of infection are being established because of obstructed drainage or retained eschars. A thorough cleansing of the wound with a two per cent solution of Liquor Cresolis Compositus and this followed by moistening every part of the wound with tincture of iodin, will check all such disturbance if done promptly.

Where practically all of the anterior surface of the radius has been denuded, recovery is tardy and there is in some cases imperfect extension of the leg for months after the wound has healed. But in such instances, animals gradually regain complete use of the affected member and in the course of a year function is fully restored.

Inflammation and Contraction of the Carpal Flexors.

Anatomy.—The structures which are usually considered as true flexors of the carpus are a group of three muscles, which have separate heads of origin and different points of tendinous insertion.

The flexor carpiradialis (flexor metacarpi internus) originates from the medial epicondyle of the humerus. It is inserted to the proximal end of the medial metacarpal (inner metacarpal or splint) bone. This muscle is the smaller of the three and is not usually divided in doing carpal tenotomy.

The flexor carpiulnaris (flexor metacarpi medius) has two heads of origin; one, the larger, originates from the epicondyle of the humerus and the other from the posterior surface of the olecranon. The two heads unite at the upper third of the radius and the muscle, becoming tendinous, as is the case with the other carpal flexors, is attached by one point of insertion to the accessory carpal bone (trapezum). The other blends with the posterior annular ligament of the carpus.

The ulnaris lateralis (flexor metacarpi externus) has its origin from the lateral epicondyle of the humerus and inserts to the proximal extremity of the fourth metacarpal (outer splint) bone and by another attachment to the accessory carpal bone (trapezium) with the tendon of the flexor carpiulnaris (flexor metacarpi medius).

Acting together, these muscles flex the carpus or extend the elbow and this action is antagonized by the biceps brachii (flexor brachii) and extensors of the carpus and phalanges.

Etiology and Occurrence.—Inflammation of the muscular or tendinous parts of the carpal flexors, does not occur as frequently as does inflammation of the flexors of the extremity. They are subject to injury such as is occasioned by hard work and concussion and contract as a result; but, more frequently a congenital malformation of the leg is responsible for undue strain upon these parts. Horses that are "knee sprung" or that have a congenital condition where in the anterior line, as formed by the radius, carpal and metacarpal bones, is bent forward at the carpus, are subject to inflammation and contraction of the carpal flexors. When these flexors are contracted, the condition is commonly known among horsemen as "buck knee." In itself, inflammation of the carpal flexors is not a condition which is likely to prove troublesome, but because of carpal involvement (which is often present) the cause of the trouble remains, and inflammation of the carpal flexors recurs or becomes chronic and contraction of tendons results.

Symptomatology.—Inflammation of the carpal flexors, when acute and uncomplicated, is characterized by a painfully swollen condition of the affected tendons. No weight is borne upon the affected leg and the carpal joint is flexed. Mixed lameness is present. There is no difficulty encountered in arriving at a diagnosis because of the very noticeably inflamed parts.

Many fully developed cases of contraction of the tendons of the carpal flexors are observed where the condition has become established gradually and no lameness has resulted from tendinitis or carpitis. In some of these cases, subjects are stumblers and when they are carelessly handled or kept at fast work over irregular or hard roads, chronic carpitis with hyperplasia of the structures of the anterior carpal region results, owing to frequent bruising from falls.



Where inflammation is caused by a puncture wound and subfascial infection occurs, there is evident manifestation of pain. No weight is supported by the affected member and because of the pressure, occasioned by the swollen muscles confined within the non-yielding brachial fascia, there exists marked supersensitiveness of the affected parts. Flexion of the elbow is avoided because contraction of the biceps brachii (flexor brachii) or the extensors, which are antagonists of the flexors of the carpus, tenses the carpal flexors and pain is thereby increased.

However, in most instances, the practitioner's attention is not directed to typical and uncomplicated cases, but to subacute or chronic inflammations which are often attended with contraction of the tendinous parts of the carpal flexors, and in such cases carpitis is present. Animals so affected have lost the rigidity which characterizes the normal carpal joint when the leg is a weight bearing member, and because of its sprung condition, the leg trembles when supporting weight.

Treatment.—Acute inflammation is treated by means of local application of cold or hot packs until the pain and acute stage of inflammation has subsided and later stimulating liniments are indicated. Absolute quiet must be enforced. Especially where the carpus is involved must the subject be kept quiet until all evidence of inflammation has subsided.

The application of vesicants or line-firing is beneficial in subacute inflammation of the tendons of the carpal flexors. Where contraction of tendons exists and no osseous or ligamentous change prevents correction of the condition, tenotomy is necessary. The reader is referred to Merillat's "Veterinary Surgery"[17] for a good description of the technic of this operation.

In all serious cases of inflammation of the carpal flexors, whether tenotomy has been performed or not, the subject needs a long period of rest subsequent to treatment. In fact, three or four months at pasture is necessary to permit of recovery and this where no congenital deformity has predisposed the subject to such affection of the flexors. Return to work must be gradual and the character of the work such as to enable the animal to become inured to service without a recurrence of the trouble if possible.

It follows then, that tenotomy, here as in other cases, is not practical from an economic viewpoint, unless the animal be of sufficient value to justify the long period of rest for recovery. Tenotomy is not of practical benefit unless ample time is allowed for regeneration of divided tendinous tissue.

Fracture and Luxation of the Carpal Bones.

Etiology and Occurrence.—Fracture of the carpal bones is of infrequent occurrence in horses and, when it does occur, it is usually due to injuries, and because of their nature (resulting as they generally do from heavy falls or in being run over by street cars or wagons), a comminuted fracture of one or more bones exists. The accessory carpal bone (trapezium) is said to be fractured at times without being subjected to blows or like injuries, but this is exceptional.

Luxations of the carpal joint are of rare occurrence, and very few cases of this kind are on record. Walters[18] reports a case of carpometacarpal luxation in a pony wherein reduction was spontaneous and an uneventful recovery followed. His reason for reporting the case, as he states, is its rarity.

Symptomatology.—Fractures of the carpal bones as they usually take place are diagnosed without difficulty. Because of their usually being comminuted, abnormal movement of the joint is possible. Such movement is not restricted and flexion of the leg at the carpus in any direction is possible. Crepitation is readily detected and frequently these fractures are of the compound-comminuted variety.

In fracture of the accessory carpal bone (trapezium) or in fracture of any other single bone when such exists, there is no increase in the movement of the joint. The accessory carpal bone may be readily manipulated and when fractured, its parts are more or less displaced. Recognition of fracture of any other single carpal bone must be done by detecting crepitation unless it be a compound fracture, whereupon probing is of aid in establishing a diagnosis.

Carpal luxation when present is to be recognized by finding the apposing carpal bones joined in an abnormal manner—that is, out of position. There is restricted or suspended function of the joint, and in the cases recorded, no difficulty has been experienced in making a diagnosis. The carpometacarpal portion of the articulation is the part which is usually affected.

Prognosis and Treatment.—There is no chance for complete recovery in the usual case of carpal fracture because of the fact that there results sufficient arthritis to destroy articular cartilage beyond repair. In the average instance, because of arthritis which persists for a considerable length of time, more or less ankylosis results. At best, one can only hope for partial recovery, that is to say, the member may regain its usefulness as a weight-supporting part, but because of restricted or abolished joint function, locomotion is more or less difficult. Exostoses, articular and periarticular, occur and the carpus usually becomes a large immobile articulation. There is danger of infection resulting in simple carpal fractures and, needless to say, in a compound-comminuted fracture of the carpus, infection usually occurs and a fatal outcome is probable.

When treatment is instituted, antiseptic precautions are taken in handling the compound fractures, and in any case immobilization of the parts is sought. Here, as has been previously pointed out, it is best to employ leather splints, so that a maximum degree of rigidity with a minimum of distress and inconvenience to the patient will result. The leg must be bandaged from the hoof upward, making use of a sufficient amount of cotton to ensure against pressure-necrosis. The leather splints are placed mesially and laterally and, of course, need to extend as high as the proximal end of the radius. Subjects must be kept in slings until union of bones has become established, and as a rule there will then exist marked ankylosis.

There is no particular difference in the handling of carpal luxation and dislocation of other bones. Where ligaments have not been destroyed to the extent that reduction is of no practical use, the parts are kept immobilized, if thought necessary. Later, vesication of the whole pericarpal region is done and the subject allowed exercise at will.

Carpitis.

Etiology and Occurrence.—Inflammation of the carpus is caused by contusions, such as are occasioned in falling, by kicks by striking the carpus against objects in jumping and sometimes by striking it against the manger in pawing. The condition is of rather frequent occurrence.

Symptomatology.—Evident symptoms of inflammation in carpitis are always present—hyperthermia, supersensitiveness and swelling. Also, there exists lameness which is characterized by an apparent inability to flex the leg, and there is circumduction of the leg as it is advanced because in this way little if any flexion of the carpus (which increases pain) is necessary.

Depending upon the nature of the cause, there occurs a marked difference in the character and amount of swelling.



Naturally, when much extravasation of serum and blood takes place, there is occasioned a fluctuating swelling which is usually less painful to the subject upon manipulation than is a dense inflammatory change without marked extravasation.

In acute carpitis, there is present, then, a very painful condition which involves the articulation, causing marked lameness, disturbance of appetite and some elevation of temperature.

Chronic cases do not occasion serious pain or constitutional disturbances, but do interfere with locomotion in direct proportion to the existing articular inflammation and periarticular hypertrophy of ligamentous and tendinous structures.

Treatment.—If possible, keep the subject absolutely quiet, employing the sling if necessary. During the first stages of inflammation, the application of ice packs to the affected parts, is of marked benefit. At the end of forty-eight hours, hot applications may be used and this treatment continued throughout several days. Anodyne liniments are of service and should be employed throughout the acute stage of inflammation during intervals between the hydrotherapeutic treatments.