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Applied Eugenics
by Paul Popenoe and Roswell Hill Johnson
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Taking an historical view, it seems fairly evident that if diseases were really inherited, the race would have been extinct long ago. Of course there are constitutional defects or abnormalities that are in the germ-plasm and are heritable: such is the peculiar inability of the blood to coagulate, which marks "bleeders" (sufferers from hemophilia, a highly hereditary disease). And in many cases it is difficult to distinguish between a real germinal condition of this sort, and an acquired disease.

The inheritance of an acquired disease is not only inconceivable, in the light of what is known about the germ-plasm, but there is no evidence to support it. While there is most decidedly such a thing as the inheritance of a tendency to or lack of resistance to a disease, it is not the result of incidence of the disease on the parent. It is possible to inherit a tendency to headaches or to chronic alcoholism; and it is possible to inherit a lack of resistance to common diseases such as malaria, small-pox or measles; but actually to inherit a zymotic disease as an inherent genetic trait, is impossible,—is, in fact, a contradiction of terms.

(3) When we come to the effects of use and disuse, we reach a much debated ground, and one complicated by the injection of a great deal of biological theorizing, as well as the presence of the usual large amount of faulty observation and inference.

It will be admitted by every one that a part of the body which is much used tends to increase in size, or strength, and similarly that a part which is not used tends to atrophy. It is further found that such changes are progressive in the race, in many cases. Man's brain has steadily increased in size, as he used it more and more; on the other hand, his canine teeth have grown smaller. Can this be regarded as the inheritance of a long continued process of use and disuse? Such a view is often taken, but the Lamarckian doctrine seems to us just as mystical here as anywhere else, and no more necessary. Progressive changes can be satisfactorily accounted for by natural selection; retrogressive changes are susceptible of explanation along similar lines. When an organ is no longer necessary, as the hind legs of a whale, for instance, natural selection no longer keeps it at the point of perfection. Variation, however, continues to occur in it. Since the organ is now useless, natural selection will no longer restrain variation in such an organ, and degeneracy will naturally follow, for of all the variations that occur in the organ, those tending to loss are more numerous than those tending to addition. If the embryonic development of a whale's hind leg be compared to some complicated mechanical process, such as the manufacture of a typewriter, it will be easier to realize that a trivial variation which affected one of the first stages of the process would alter all succeeding stages and ruin the final perfection of the machine. It appears, then, that progressive degeneration of an organ can be adequately explained by variation with the removal of natural selection, and that it is not necessary or desirable to appeal to any Lamarckian factor of an unexplainable and undemonstrable nature.

The situation remains the same, when purely mental processes, such as instincts, are considered. Habit often repeated becomes instinctive, it is said; and then the instinct thus formed by the individual is passed on to his descendants and becomes in the end a racial instinct. Most psychologists have now abandoned this view, which receives no support from investigation. Such prevalence as it still retains seems to be largely due to a confusion of thought brought about by the use of the word "instinctive" in two different senses,—first literally and then figuratively.

A persistent attempt has been made in America during recent years, by C. L. Redfield, a Chicago engineer, to rehabilitate the theory of the inheritance of the effects of use and disuse. He has presented it in a way that, to one ignorant of biology, appears very exact and plausible; but his evidence is defective and his interpretation of his evidence fallacious. Because of the widespread publicity, Mr. Redfield's work has received, we discuss it further in Appendix B.

Since the importance of hormones (internal secretions) in the body became known, it has often been suggested that their action may furnish the clue to some sort of an inheritance of modifications. The hormone might conceivably modify the germ-plasm but if so, it would more likely be in some wholly different way.

In general, we may confidently say that there is neither theoretical necessity nor adequate experimental proof for belief that the results of use and disuse are inherited.

(4) When we come to consider whether the effects of the environment are inherited, we attack a stronghold of sociologists and historians. Herbert Spencer thought one of the strongest pieces of evidence in this category was to be found in the assimilation of foreigners in the United States. "The descendants of the immigrant Irish," he pointed out, "lose their Celtic aspect and become Americanised.... To say that 'spontaneous variation,' increased by natural selection, can have produced this effect, is going too far." Unfortunately for Mr. Spencer, he was basing his conclusions on guesswork. It is only within the last few months that the first trustworthy evidence on the point has appeared, in the careful measurements of Hrdlicka who has demonstrated that Spencer was quite wrong in his statement. As a fact, the original traits persist with almost incredible fidelity. (Appendix C.)

In 1911, Franz Boas of Columbia University published measurements of the head form of children of immigrants[14] which purported to show that American conditions caused in some mysterious manner a change in the shape of the head. This conclusion in itself would have been striking enough, but was made more startling when he announced that the change worked both ways: "The East European Hebrew, who has a very round head, becomes more long-headed; the south Italian, who in Italy has an exceedingly long head, becomes more short-headed"; and moreover this potent influence was alleged to be a subtle one "which does not affect the young child born abroad and growing up in American environment, but which makes itself felt among the children born in America, even a short time after the arrival of the parents in this country." Boas' work was naturally pleasing to sociologists who believe in the reality of the "melting-pot," and has obtained widespread acceptance in popular literature. It has obtained little acceptance among his fellow-anthropologists, some of whom allege that it is unsound because of the faulty methods by which the measurements were made and the incorrect standards used for comparison.

The many instances quoted by historians, where races have changed after immigration, are to be explained in most cases by natural selection under new conditions, or by interbreeding with the natives, and not as the direct result of climate. Ellsworth Huntington, the most recent and careful student of the effect of climate on man,[15] finds that climate has a great deal of influence on man's energy, but as far as inherited traits in general are concerned, he is constantly led to remark how little heredity is capable of being changed.

Most members of the white race have little toes that are partly atrophied, and considerably deformed. In many cases one of the joints has undergone ankylosis—that is, the bones have coalesced. It is confidently alleged that this is due to the inheritance of the effects of wearing tight shoes through many centuries. When it is found that the prehistoric Egyptians, who knew not tight shoes, suffered from the same defect in a similar degree, one's confidence in this kind of evidence is much diminished.

The retrogression of the little toe in man is probably to be explained like the degeneration of the hind leg of the whale, as a result of the excess of deteriorating variations which, when not eliminated by natural selection, lead to atrophy. Since man began to limit the use of his feet to walking on the ground, the little toe has had much less value to him.

The feet of Chinese women offer another illustration along this line. Although they have been tightly bound for many generations, no deformity is apparent in the feet of girl babies.

Breeders are generally of the opinion that good care and feed bestowed on their stock produce results in succeeding generations. This is in a way true, but it is due merely to the fact that the offspring get better nourishment and therefore a better start in life. The changes in breeds, the increase in milk yield, and similar facts, often explained as due to inheritance of acquired characters, are better explained as the results of selection, sometimes conscious, sometimes quite unconscious.



The question of inherited immunity to diseases, as the result of vaccination or actual illness from them, has appeared in the controversy in a number of forms, and is a point of much importance. It is not yet clear, partly because the doctors disagree as to what immunity is. But there is no adequate evidence that an immunity to anything can be created and transmitted through the germ-plasm to succeeding generations.

In short, no matter what evidence we examine, we must conclude that inheritance of acquired bodily characters is not a subject that need be reckoned with, in applied eugenics.

On the other hand, there is a possible indirect influence of modifications, which may have real importance in man. If the individual is modified in a certain way, in a number of generations, even though such a modification is not transmitted to his descendants, yet its continued existence may make possible, the survival of some germinal variation bearing in the same direction, which without the protecting influence of the pre-existing modification, would have been swamped or destroyed.

Finally, it should be borne in mind that even if physical and mental characters acquired during a man's lifetime are not transmitted, yet there is a sort of transmission of acquired characters which has been of immense importance to the evolution of the race. This is the so-called "inheritance" of the environment; the passing on from one generation to the next of the achievements of the race, its accumulated social experience; its civilization, in short. It is doubtful whether any useful end is gained by speaking of this continuance of the environment as "heredity;" it certainly tends to confuse many people who are not used to thinking in biological terms. Tradition is the preferable term.

There is much to be said in favor of E. B. Poulton's definition,—"Civilization in general is the sum of those contrivances which enable human beings to advance independently of heredity." Whatever wisdom, material gain, or language is acquired by one generation may be passed on to the next. As far as the environment is concerned, one generation stands on the shoulders of its predecessor. It might simplify the task of eugenics if the same could be said of biological heredity. But it can not. Each generation must "start from scratch."

In August Weismann's words, the development of a function in offspring begins at the point where it began in his parents, not at the point where it ended in them. Biological improvement of the race (and such improvement greatly fosters all other kinds) must be made through a selective birth-rate. There is no short-cut by way of euthenics, merely.

We must now consider whether there is any direct way of impairing good heredity. It is currently believed that there are certain substances, popularly known as "racial-poisons," which are capable of affecting the germ-plasm adversely and permanently in spite of its isolation and protection. For example, the literature of alcoholism, and much of the literature of eugenics, abounds with statements to the effect that alcohol originates degeneracy in the human race.

The proof or disproof of this proposition must depend in the last analysis on direct observation and carefully controlled experiments. As the latter cannot be made feasibly on man, a number of students have taken up the problem by using small animals which are easily handled in laboratories. Many of these experiments are so imperfect in method that, when carefully examined, they are found to possess little or no value as evidence on the point here discussed.

Hodge, Mairet and Combemale, for example, have published data which convinced them that the germ-plasm of dogs was injured by the administration of alcohol. The test was the quality of offspring directly produced by the intoxicated animals under experiment. But the number of dogs used was too small to be conclusive, and there was no "control": hence these experiments carry little weight.

Ovize, Fere and Stockard have shown that the effect of alcohol on hen's eggs is to produce malformed embryos. This, however, is a case of influencing the development of the individual, rather than the germ-plasm. Evidence is abundant that individual development can be harmed by alcohol, but the experiments with eggs are not to the point of our present purpose.

Carlo Todde and others have carried out similar experiments on cocks. The conclusions have in general been in favor of injury to the germ-plasm, but the experiments were inadequate in extent.

Laitinen experimented on rabbits and guinea pigs, but he used small doses and secured only negative results.

Several series of experiments with rats indicate that if the dosage is large enough, the offspring can be affected.

Nice, using very small numbers of white mice, subjected them not only to alcohol, but to caffein, nicotin, and tobacco smoke. The fecundity of all these sets of mice was higher than that of the untreated ones used as control; all of them gained in weight; of 707 young, none was deformed, none stillborn, and there was only one abortion. The young of the alcoholized mice surpassed all others in growth. The dosage Nice employed was too small, however, to give his experiment great weight.

At the University of Wisconsin, Leon J. Cole has been treating male rabbits with alcohol and reports that "what appear to be decisive results have already been obtained. In the case of alcoholic poisoning of the male the most marked result has been a lessening of his efficiency as a sire, the alcohol apparently having had some effect on the vitality of his spermatozoa." His experiment is properly planned and carried out, but so far as results have been made public, they do not appear to afford conclusive evidence that alcohol originates degeneracy in offspring.

The long-continued and carefully conducted experiment of Charles R. Stockard at the Cornell Medical College is most widely quoted in this connection. He works with guinea-pigs. The animals are intoxicated daily, six days in the week, by inhaling the fumes of alcohol to the point where they show evident signs of its influence; their condition may thus be compared to that of the toper who never gets "dead drunk" but is never entirely sober. Treatment of this sort for a period as long as three years produces no apparent bad effect on the individuals; they continue to grow and become fat and vigorous, taking plenty of food and behaving in a normal manner in every particular. Some of them have been killed from time to time, and all the tissues, including the reproductive glands, have been found perfectly normal. "The treated animals are, therefore, little changed or injured so far as their behavior and structure goes. Nevertheless, the effects of the treatment are most decidedly indicated by the type of offspring to which they give rise, whether they are mated together or with normal individuals."

Before the treatment is begun, every individual is mated at least once, to demonstrate its possibility of giving rise to sound offspring. The crucial test of the influence of alcohol on the germ-cells is, of course, the mating of a previously alcoholized male with a normal, untreated female, in a normal environment.

When the experiment was last reported,[16] it had covered five years and four generations. The records of 682 offspring produced by 571 matings were tabulated, 164 matings of alcoholized animals, in which either the father, mother, or both were alcoholic, gave 64, or almost 40%, negative results or early abortions, while only 25% of the control matings failed to give full-term litters. Of the 100 full-term litters from alcoholic parents 18% contained stillborn young and only 50% of all the matings resulted in living litters, while 47% of the individuals in the litters of living young died soon after birth. In contrast to this record 73% of the 90 control matings gave living litters and 84% of the young in these litters survived as normal, healthy animals.

"The mating records of the descendants of the alcoholized guinea pigs, although they themselves were not treated with alcohol, compare in some respects even more unfavorably with the control records than do the above data from the directly alcoholized animals." The records of the matings in the second filial generation "are still worse, higher mortality and more pronounced deformities, while the few individuals which have survived are generally weak and in many instances appear to be quite sterile even though paired with vigorous, prolific, normal mates."

We do not minimize the value of this experiment, when we say that too much weight has been popularly placed on its results. Compare it with the experiment with fowls at the University of Maine, which Raymond Pearl reports.[17] He treated 19 fowls with alcohol, little effect on the general health being shown, and none on egg production. From their eggs 234 chicks were produced; the average percentage of fertility of the eggs was diminished but the average percentage of hatchability of fertile eggs was increased. The infant mortality of these chicks was smaller than normal, the chicks were heavier when hatched and grew more rapidly than normal afterwards. No deformities were found. "Out of 12 different characters for which we have exact quantitative data, the offspring of treated parents taken as a group are superior to the offspring of untreated parents in 8 characters," in two characters they are inferior and in the remaining two there is no discernible difference. At this stage Dr. Pearl's experiment is admittedly too small, but he is continuing it. As far as reported, it confirms the work of Professor Nice, above mentioned, and shows that what is true for guinea pigs may not be true for other animals, and that the amount of dosage probably also makes a difference. Dr. Pearl explains his results by the hypothesis that the alcohol eliminated the weaker germs in the parents, and allowed only the stronger germs to be used for reproduction.

Despite the unsatisfactory nature of much of the alleged evidence, we must conclude that alcohol, when given in large enough doses, may sometimes affect the germ-plasm of some lower animals in such a way as to deteriorate the quality of their offspring. This effect is probably an "induction," which does not produce a permanent change in the bases of heredity, but will wear away in a generation or two of good surroundings. It must be remembered that although the second-generation treated males of Dr. Stockard's experiment produced defective offspring when mated with females from similarly treated stock, they produced normal offspring when mated with normal females. The significance of this fact has been too little emphasized in writings on "racial poisons." If a normal mate will counteract the influence of a "poisoned" one, it is obvious that the probabilities of danger to any race from this source are much decreased, while if only a small part of the race is affected, and mates at random, the racial damage might be so small that it could hardly be detected.

There are several possible explanations of the fact that injury is found in some experiments but not in others. It may be, as Dr. Pearl thinks, that only weak germs are killed by moderate treatment, and the strong ones are uninjured. And it is probable (this applies more particularly to man) that the body can take care of a certain amount of alcohol without receiving any injury therefrom; it is only when the dosage passes the "danger point" that the possibility of injury appears. As to the location of this limit, which varies with the species, little is known. Much more work is needed before the problem will be fully cleared up.

Alcohol has been in use in parts of the world for many centuries; it was common in the Orient before the beginning of historical knowledge. Now if its use by man impairs the germ-plasm, then it seems obvious that the child of one who uses alcohol to a degree sufficient to impair his germ-plasm will tend to be born inferior to his parent. If that child himself is alcoholic, his own offspring will suffer still more, since they must carry the burden of two generations of impairment. Continuing this line of reasoning over a number of generations, in a race where alcohol is freely used by most of the population, one seems unable to escape from the conclusion that the effects of this racial poison, if it be such, must necessarily be cumulative. The damage done to the race must increase in each generation. If the deterioration of the race could be measured, it might even be found to grow in a series of figures representing arithmetical progression.

It seems impossible, with such a state of affairs, that a race in which alcohol was widely used for a long period of time, could avoid extinction. At any rate, the races which have used alcohol longest ought to show great degeneracy—unless there be some regenerative process at work constantly counteracting this cumulative effect of the racial poison in impairing the germ-plasm.

Such a proposition at once demands an appeal to history. What is found in examination of the races that have used alcohol the longest? Have they undergone a progressive physical degeneracy, as should be expected?

By no means. In this particular respect they seem to have become stronger rather than weaker, as time went on; that is, they have been less and less injured by alcohol in each century, as far as can be told. Examination of the history of nations which are now comparatively sober, although having access to unlimited quantities of alcohol, shows that at an earlier period in their history, they were notoriously drunken; and the sobriety of a race seems to be proportioned to the length of time in which it has had experience of alcohol. The Mediterranean peoples, who have had abundance of it from the earliest period recorded, are now relatively temperate. One rarely sees a drunkard among them, although many individuals in them would never think of drinking water or any other non-alcoholic beverage. In the northern nations, where the experience of alcohol has been less prolonged, there is still a good deal of drunkenness, although not so much as formerly. But among nations to whom strong alcohol has only recently been made available—the American Indian, for instance, or the Eskimo—drunkenness is frequent wherever the protecting arm of government does not interfere.

What bearing does this have on the theory of racial poisons?

Surely a consideration of the principle of natural selection will make it clear that alcohol is acting as an instrument of racial purification through the elimination of weak stocks. It is a drastic sort of purification, which one can hardly view with complacency; but the effect, nevertheless, seems clear cut.

To demonstrate the action of natural selection, we must first demonstrate the existence of variations on which it can act. This is not difficult in the character under consideration—namely, the greater or less capacity of individuals to be attracted by alcohol, to an injurious degree.

As G. Archdall Reid has pointed out,[18] men drink for at least three different reasons: (1) to satisfy thirst. This leads to the use of a light wine or a malt liquor. (2) To gratify the palate. This again usually results in the use of drinks of low alcohol content, in which the flavor is the main consideration. (3) Finally, men drink "to induce those peculiar feelings, those peculiar frames of mind" caused by alcohol.

Although the three motives may and often do coexist in the same individual, or may animate him at different periods of life, the fact remains that they are quite distinct. Thirst and taste do not lead to excessive drinking; and there is good evidence that the degree of concentration and the dosage are important factors in the amount of harm alcohol may do to the individual. The concern of evolutionists, therefore, is with the man who is so constituted that the mental effects of alcohol acting directly on the brain are pleasing, and we must show that there is a congenital variability in this mental quality, among individuals.

Surely an appeal to personal experience will leave little room for doubt on that point. The alcohol question is so hedged about with moral and ethical issues that those who never get drunk, or who perhaps never even "take a drink," are likely to ascribe that line of conduct to superior intelligence and great self-control. As a fact, a dispassionate analysis of the case will show that why many such do not use alcoholic beverages to excess is because intoxication has no charm for them. He is so constituted that the action of alcohol on the brain is distasteful rather than pleasing to him. In other cases it is variation in controlling satisfaction of immediate pleasures for later greater good.

Some of the real inebriates have a strong will and a real desire to be sober, but have a different mental make-up, vividly described by William James:[19] "The craving for drink in real dipsomaniacs, or for opium and chloral in those subjugated, is of a strength of which normal persons can have no conception. 'Were a keg of rum in one corner of the room, and were a cannon constantly discharging balls between me and it, I could not refrain from passing before that cannon in order to get that rum. If a bottle of brandy stood on one hand, and the pit of hell yawned on the other, and I were convinced I should be pushed in as surely as I took one glass, I could not refrain.' Such statements abound in dipsomaniacs' mouths." Between this extreme, and the other of the man who is sickened by a single glass of beer, there are all intermediates.

Now, given an abundant and accessible supply of alcohol to a race, what happens? Those who are not tempted or have adequate control, do not drink to excess; those who are so constituted as to crave the effects of alcohol (once they have experienced them), and who lack the ability to deny themselves the immediate pleasure for the sake of a future gain, seek to renew these pleasures of intoxication at every opportunity; and the well attested result is that they are likely to drink themselves to a premature death.

Although it is a fact that the birth-rate in drunkard's families may be and often is larger than that of the general population,[20] it is none the less a fact that many of the worst drunkards leave no or few offspring. They die of their own excesses at an early age; or their conduct makes them unattractive as mates; or they give so little care to their children that the latter die from neglect, exposure or accident. As these drunkards would tend to hand down their own inborn peculiarity, or weakness for alcohol, to their children, it must be obvious that their death results in a smaller proportion of such persons in the next generation. In other words, natural selection is at work again here, with alcohol as its agent. By killing off the worst drunkards in each generation, nature provides that the following generation shall contain fewer people who lack the power to resist the attraction of the effect of alcohol, or who have a tendency to use it to such an extent as to injure their minds and bodies. And it must be obvious that the speed and efficacy of this ruthless temperance reform movement are proportionate to the abundance and accessibility of the supply of alcohol. Where the supply is ample and available, there is certain to be a relatively high death-rate among those who find it too attractive, and the average of the race therefore is certain to become stronger in this respect with each generation. Such a conclusion can be abundantly justified by an appeal to the history of the Teutonic nations, the nations around the Mediterranean, the Jews, or any race which has been submitted to the test.

There seems hardly room for dispute on the reality of this phase of natural selection. But there is another way in which the process of strengthening the race against the attraction and effect of alcohol may be going on at the same time. If the drug does actually injure the germ-plasm, and set up a deterioriation, it is obvious that natural selection is given another point at which to work. The more deteriorated would be eliminated in each generation in competition with the less deteriorated or normal; and the process of racial purification would then go on the more rapidly. The fact that races long submitted to the action of alcohol have become relatively resistant to it, therefore, does not in itself answer the question of whether alcohol injures the human germ-plasm.

The possible racial effect of alcoholization is, in short, a much more complicated problem than it appears at first sight to be. It involves the action of natural selection in several important ways, and this action might easily mask the direct action of alcohol on the germ-plasm, if there be any measurable direct result.

No longer content with a long perspective historical view, we will scrutinize the direct investigations of the problem which have been made during recent years. These investigations have in many cases been widely advertised to the public, and their conclusions have been so much repeated that they are often taken at their face value, without critical examination.

It must be borne in mind that the solution of the problem depends on finding evidence of degeneracy or impairment in the offspring of persons who have used alcohol, and that this relation might be explainable in one or more of three ways:

(1) It may be that alcoholism is merely a symptom of a degenerate stock. In this case the children will be defective, not because their parents drank, but because their parents were defective—the parents' drinking being merely one of the symptoms of their defect.

(2) It may be that alcohol directly poisons the germ-plasm, in such a way that parents of sound stock, who drink alcoholic beverages, will have defective offspring.

(3) It may be that the degeneracy observed in the children of drunkards (for of course no one will deny that children of drunkards are frequently defective) is due solely to social and economic causes, or other causes in the environment: that the drunken parents, for instance, do not take adequate care of their children, and that this lack of care leads to the defects of the children.

The latter influence is doubtless one that is nearly always at work, but it is wholly outside the scope of the present inquiry, and we shall therefore ignore it, save as it may appear incidentally. Nor does it require emphasis here; for the disastrous social and economic effects of alcoholism are patent to every observer. We find it most convenient to concentrate our attention first on the second of the questions above enumerated: to ask whether there is any good evidence that the use of alcoholic beverages by men and women really does originate degeneracy in their offspring.

To get such evidence, one must seek an instance that will be crucial, one that will leave no room for other interpretations. One must, therefore, exclude consideration of cases where a mother drank before child birth. It is well-known that alcohol can pass through the placenta, and that if a prospective mother drinks, the percentage of alcohol in the circulation of the unborn child will very soon be nearly equal to that in her own circulation. It is well established that such a condition is extremely injurious to the child; but it has nothing directly to do with heredity. Therefore we can not accept evidence of the supposed effect of alcohol on the fertilized egg-cell, at any stage in its development, because that is an effect on the individual, not on posterity. And the only means by which we can wholly avoid this fallacy is to give up altogether an attempt to prove our case by citing instances in which the mother was alcoholic. If this is not done, there will always be liability of mistaking an effect of prenatal nutrition for a direct injury to the germ-plasm.

But if we can find cases where the mother was of perfectly sound stock, and non-alcoholic; where the father was of sound stock, but alcoholic; and where the offspring were impaired in ways that can be plausibly attributed to an earlier injury to the germ-plasm by the father's alcohol; then we have evidence that must weigh heavily with the fair-minded.

An interesting case is the well-known one recorded by Schweighofer, which is summarized as follows: "A normal woman married a normal man and had three sound children. The husband died and the woman married a drunkard and gave birth to three other children; one of these became a drunkard; one had infantilism, while the third was a social degenerate and a drunkard. The first two of these children contracted tuberculosis, which had never before been in the family. The woman married a third time and by this sober husband again produced sound children."

Although such evidence is at first sight pertinent, it lacks much of being convincing. Much must be known about the ancestry of the drunken husband, and of the woman herself, before it can be certain that the defective children owe their defect to alcoholism rather than to heredity.

We can not undertake to review all the literature of this subject, for it fills volumes, but we shall refer to a few of the studies which are commonly cited, by the believers in the racial-poison character of alcohol, as being the most weighty.

Taav Laitinen of Helsingfors secured information from the parents of 2,125 babies, who agreed to weigh their infants once a month for the first eight months after birth, and who also furnished information about their own drinking habits. His conclusion is that the average weight of the abstainer's child is greater at birth, that these children develop more rapidly during the first eight months than do the children of the moderate drinker, and that the latter exceed in the same way the children of the heavier drinker. But a careful analysis of his work by Karl Pearson, whose great ability in handling statistics has thrown light on many dark places in the alcohol problem, shows[21] that Professor Laitinen's statistical methods were so faulty that no weight can be attached to his conclusions. Furthermore, he appears to have mixed various social classes and races together without distinction; and he has made no distinction between parents, one of whom drank, and parents, both of whom drank. Yet, this distinction, as we have pointed out, is a critical one for such inquiries. Professor Laitinen's paper, according to one believer in racial poisons, "surpasses in magnitude and precision all the many studies of this subject which have proved the relation between drink and degeneracy." As a fact, it proves nothing of the sort as to race degeneracy.

Again, T. A. MacNicholl reported on 55,000 American school children, from 20,147 of whom he secured information about the parents' attitude to alcoholic drinks. He found an extraordinarily large proportion (58%) of deficient and backward children in the group. But the mere bulk of his work, probably, has given it far more prestige than it deserves; for his methods are careless, his classifications vague, his information inadequate; he seems to have dealt with a degenerate section of the population, which does not offer suitable material for testing the question at issue; and he states that many of the children drank and smoked,—hence, any defects found in them may be due to their own intemperance, rather than that of their parents. In short, Dr. MacNicholl's data offer no help in an attempt to decide whether alcoholism is an inheritable effect.

Another supposed piece of evidence which has deceived a great many students is the investigation of Bezzola into the distribution of the birth-rate of imbeciles in Switzerland. He announced that in wine-growing districts the number of idiots conceived at the time of the vintage and carnival is very large, while at other periods it is almost nil. The conclusion was that excesses of drunkenness occurring in connection with the vintage and carnival caused this production of imbeciles. But aside from the unjustified assumptions involved in his reasoning, Professor Pearson has recently gone over the data and shown the faulty statistical method; that, in fact, the number of imbeciles conceived at vintage-time, in excess of the average monthly number, was only three in spite of the large numbers! Bezzola's testimony, which has long been cited as proof of the disastrous results of the use of alcohol at the time of conception, must be discarded.

Demme's plausible investigation is also widely quoted to support the belief that alcohol poisons the germ-plasm. He studied the offspring of 10 drunken and 10 sober pairs of parents, and found that of the 61 children of the latter, 50 were normal, while of the 57 progeny of the drunkards, only nine were normal. This is a good specimen of much of the evidence cited to prove that alcohol impairs the germ-plasm; it has been widely circulated by propagandists in America during recent years. Of course, its value depends wholly on whether the 20 pairs of parents were of sound, comparable stock. Karl Pearson has pointed out that this is not the case. Demme selected his children of drunkards by selecting children who came to his hospital on account of imperfect development of speech, mental defect, imbecility or idiocy. When he found families in which such defective children occurred, he then inquired as to their ancestry. Many of these children, he found, were reduced to a condition approaching epilepsy, or actually epileptic, because they themselves were alcoholic. Obviously such material can not legitimately be used to prove that the use of alcohol by parents injures the heredity of their children. The figures do not at all give the proof we are seeking, that alcohol can so affect sound germ-plasm as to lead to the production of defective children.

Dr. Bertholet made a microscopic examination of the reproductive glands of 75 chronic male alcoholics, and in 37 cases he found them more or less atrophied, and devoid of spermatozoa. Observing the same glands in non-alcoholics who had died of various chronic diseases, such as tuberculosis, he found no such condition. His conclusion is that the reproductive glands are more sensitive to the effects of alcohol than any other organ. So far as is known to us, his results have never been discredited; they have, on the contrary, been confirmed by other investigators. They are of great significance to eugenics, in showing how the action of natural selection to purge the race of drunkards is sometimes facilitated in a way we had not counted, through reduced fertility due to alcohol, as well as through death due to alcohol. But it should not be thought that his results are typical, and that all chronic alcoholists become sterile: every reader will know of cases in his own experience, where drunkards have large families; and the experimental work with smaller animals also shows that long-continued inebriety is compatible with great fecundity. It is probable that extreme inebriety reduces fertility, but a lesser amount increases it in the cases of many men by reducing the prudence which leads to limited families.

In 1910 appeared the investigation of Miss Ethel M. Elderton and Karl Pearson on school children in Edinburgh and Manchester.[22] Their aim was to take a population under the same environmental conditions, and with no discoverable initial differentiation, and inquire whether the temperate and intemperate sections had children differing widely in physique and mentality. Handling their material with the most refined statistical methods, and in an elaborate way, they reached the conclusion that parental alcoholism does not markedly affect the physique or mentality of the offspring as children. Whether results might differ in later life, their material did not show. Their conclusions were as follows:

"(1) There is a higher death-rate among the offspring of alcoholic than among the offspring of sober parents. This appears to be more marked in the case of the mother than in the case of the father, and since it is sensibly higher in the case of the mother who has drinking bouts [periodical sprees] than of the mother who habitually drinks, it would appear to be due very considerably to accidents and gross carelessness and possibly in a minor degree to toxic effect on the offspring.

"Owing to the greater fertility of alcoholic parents, the net family of the sober is hardly larger than the net family of the alcoholic. [It should be remembered that the study did not include childless couples.]

"(2) The mean weight and height of the children of alcoholic parents are slightly greater than those of sober parents, but as the age of the former children is slightly greater, the correlations when corrected for age are slightly positive, i.e., there is slightly greater height and weight in the children of the sober."

"(3) The wages of the alcoholic as contrasted with the sober parent show a slight difference compatible with the employers' dislike for an alcoholic employee, but wholly inconsistent with a marked mental or physical inferiority in the alcoholic parent.

"(4) The general health of the children of alcoholic parents appears on the whole slightly better than that of sober parents. There are fewer delicate children, and in a most marked way cases of tuberculosis and epilepsy are less frequent than among the children of sober parents. The source of this relation may be sought in two directions; the physically strongest in the community have probably the greatest capacity and taste for alcohol. Further the higher death rate of the children of alcoholic parents probably leaves the fittest to survive. Epilepsy and tuberculosis both depending upon inherited constitutional conditions, they will be more common in the parents of affected offspring, and probably if combined with alcohol, are incompatible with any length of life or size of family. If these views be correct, we can only say that parental alcoholism has no marked effect on filial health.

"(5) Parental alcoholism is not the source of mental defect in offspring.

"(6) The relationship, if any, between parental alcoholism and filial intelligence is so slight that even its sign can not be determined from the present material.

"(7) The normal visioned and normal refractioned offspring appear to be in rather a preponderance in the families of the drinking parents, the parents who have 'bouts' give intermediate results, but there is no substantial relationship between goodness of sight and parental alcoholism. Some explanation was sought on the basis of alcoholic homes driving the children out into the streets. This was found to be markedly the case, the children of alcoholic parents spending much more of their spare time in the streets. An examination, however, of the vision and refraction of children with regard to the time they spent in-and out-of-doors, showed no clear and definite result, the children who spent the whole or most of their spare time in the streets having the most myopia and also most normal sight. It was not possible to assert that the outdoor life was better for the sight, or that the better sight of the offspring of alcoholic parentage was due to the greater time spent outdoors.

"(8) The frequency of diseases of the eye and eyelids, which might well be attributed to parental neglect, was found to have little, if any, relation to parental alcoholism.

"To sum up, then no marked relation has been found between the intelligence, physique or disease of the offspring and the parental alcoholism in any of the categories mentioned. On the whole the balance turns as often in favor of the alcoholic as of the non-alcoholic parentage. It is needless to say that we do not attribute this to the alcohol but to certain physical and possibly mental characters which appear to be associated with the tendency to alcohol."

Of the many criticisms made of this work, most are irrelevant to our present purpose, or have been satisfactorily met by the authors. It must be said, however, that as the children examined were all school children, the really degenerate offspring of alcoholics, if any such existed, would not have been found, because they would not have been admitted to the school. Further, it is not definitely known whether the parents' alcoholism dated from before or after the birth of the child examined. Then, the report did not exactly compare the offspring of drinkers and non-drinkers, but classified the parents as those who drank, and those who were sober; the latter were not, for the most part, teetotalers, but merely persons whose use of alcohol was so moderate that it exercised no visible bad influence on the health of the individual or the welfare of the home. Something can be said on both sides of all these objections; but giving them as much weight as one thinks necessary, the fact remains that the Elderton-Pearson investigation failed to demonstrate any racial poisoning due to alcohol, in the kind of cases where one would certainly have expected it to be demonstrated, if it existed.

Much more observation and measurement must be made before a generalization can be safely drawn, as to whether alcohol is or is not a racial poison, in the sense in which that expression is used by eugenists. It has been shown that the evidence which is commonly believed to prove beyond doubt that alcohol does injure the germ-plasm, is mostly worthless. But it must not be thought that the authors intend to deny that alcohol is a racial poison, where the dosage is very heavy and continuous. If we have no good evidence that it is, we equally lack evidence on the other side. We wish only to suggest caution against making rash generalizations on the subject which lack supporting evidence and therefore are a weak basis for propaganda.

So far as immediate action is concerned, eugenics must proceed on the basis that there is no proof that alcohol as ordinarily consumed will injure the human germ-plasm. To say this is not in any way to minify the evil results which alcohol often has on the individual, or the disastrous consequences to his offspring, euthenically. But nothing is to be gained by making an assumption of "racial poisoning," and acting on that assumption, without evidence that it is true; and the temperance movement would command more respect from genetics if it ceased to allege proof that alcohol has a directly injurious effect on the race, by poisoning the human germ-plasm, when no adequate proof exists.

How, then, can one account for the immense bulk of cases, some of which come within everyone's range of vision, where alcoholism in the parent is associated with defect in the offspring? By a process of exclusion, we are driven to the explanation already indicated: that alcoholism may be a symptom, rather than a cause, of degeneracy. Some drunkards are drunkards, because they come of a stock that is, in a way, mentally defective; physical defects are frequently correlated in such stocks; naturally the children inherit part or all of the parental defects including, very likely, alcoholism; but the parent's alcoholism, we repeat, must not be considered the cause of the child's defect. The child would have been defective in the same way, regardless of the parent's beverage.

It follows, then, as a practical consequence for eugenics, that in the light of present knowledge any campaign against alcoholic liquors would be better based on the very adequate ground of physiology and economics, than on genetics. From the narrowest point of view of genetics, the way to solve the liquor problem would be, not to eliminate drink, but to eliminate the drinker: to prevent the reproduction of the degenerate stocks and the tainted strains that contribute most of the chronic alcoholics. We do not mean to advocate this as the only proper basis for the temperance campaign, because the physiological and economic aspects are of sufficient importance to keep up the campaign at twice the present intensity.[23] But it is desirable to have the eugenic aspect of the matter clearly understood, and to point out that in checking the production of defectives in the United States, eugenics will do its share, and a big share, toward the solution of the drink problem, which is at the same time being attacked along other and equally praiseworthy lines by other people.

A number of other substances are sometimes credited with being racial poisons.

The poison of Spirochaete pallida, the microoerganism which causes syphilis, has been widely credited with a directly noxious effect on the germ-plasm, and the statement has been made that this effect can be transmitted for several generations. On the other hand, healthy children are reported as being born to cured syphilitics. Further evidence is needed, taking care to eliminate cases of infection from the parents. If the alleged deterioration really occurs, it will still remain to be determined if the effect is permanent or an induction, that is, a change in the germ-cells which does not permanently alter the nature of the inherited traits, and which would disappear in a few generations under favorable conditions.

The case against lead is similar. Sir Thomas Oliver, in his Diseases of Occupation, sums up the evidence as follows:

"Rennert has attempted to express in statistical terms the varying degrees of gravity in the prognosis of cases in which at the moment of conception both parents are the subjects of lead poisoning, also when one alone is affected. The malign influence of lead is reflected upon the fetus and upon the continuation of the pregnancy 94 times out of 100 when both parents have been working in lead, 92 times when the mother alone is affected, and 63 times when it is the father alone who has worked in lead. Taking seven healthy women who were married to lead workers, and in whom there was a total of 32 pregnancies, Lewin (Berlin) tells us that the results were as follows: 11 miscarriages, one stillbirth, 8 children died within the first year after their birth, four in the second year, five in the third year and one subsequent to this, leaving only two children out of 32 pregnancies as likely to live to manhood. In cases where women have had a series of miscarriages so long as their husbands worked in lead, a change of industrial occupation on the part of the husband restores to the wives normal child-bearing powers." The data of Constantin Paul, published as long ago as 1860, indicated that lead exercised an injurious effect through the male as well as the female parent. This sort of evidence is certainly weak, in that it fails to take into account the possible effects of environment; and one would do well to keep an open mind on the subject. In a recent series of careful experiments at the University of Wisconsin, Leon J. Cole has treated male rabbits with lead. He reports: "The 'leaded' males have produced as many or more offspring than normal fathers, but their young have averaged smaller in size and are of lowered vitality, so that larger numbers of them die off at an early age than is the case with those from untreated fathers."

[Illustration: EFFECT OF LEAD AS A "RACIAL POISON"

FIG. 7.—That lead poisoning can affect the germ plasm of rabbits is indicated by experiments conducted by Leon J. Cole at the University of Wisconsin. With reference to the above illustration, Professor Cole writes: "Each of the photographs shows two young from the same litter, in all cases the mother being a normal (nonpoisoned) albino. In each of the litters the white young is from an albino father which received the lead treatment, while the pigmented offspring is from a normal, homozygous, pigmented male. While these are, it is true, selected individuals, they represent what tend to be average, rather than extreme, conditions. The albino male was considerably larger than the pigmented male; nevertheless his young average distinctly smaller in size. Note also the brighter expression of the pigmented young."]

There is, then, a suspicion that lead is a racial poison, but no evidence as yet as to whether the effect is permanent or in the nature of an induction.

This concludes the short list of substances for which there has been any plausible case made out, as racial poisons. Gonorrhea, malaria, arsenic, tobacco, and numerous other substances have been mentioned from time to time, and even ardently contended by propagandists to be racial poisons, but in the case of none of them, so far as we know, is there any evidence to support the claim. And as has been shown, in the case of the three chief so-called racial poisons, alcohol, syphilis and lead, the evidence is not great. We are thus in a position to state that, from the eugenists' point of view, the origination of degeneracy, by some direct action of the germ-plasm, is a contingency that hardly needs to be reckoned with. Even in case the evidence were much stronger than it is, the damage done may only be a physiological or chemical induction, the effects of which will wear off in a few generations; rather than a radical change in the hereditary constituents of the germ-plasm. The germ-plasm is so carefully isolated and guarded that it is almost impossible to injure it, except by treatment so severe as to kill it altogether; and the degeneracy with which eugenists are called on to deal is a degeneracy which is running along from generation to generation and which, when once stopped by the cessation of reproduction, is in little danger of being originated anew through some racial poison.

Through these facts, the problem of race betterment is not only immensely simplified, but it is clearly shown to be more a matter for treatment by the biologist, acting through eugenics, than for the optimistic improver of the environment.

There is another way in which it is widely believed that some such result as a direct influence of the germ-plasm can be produced: that is through the imaginary process known as maternal impression, prenatal influence, etc. Belief in maternal impressions is no novelty. In the book of Genesis[24] Jacob is described as making use of it to get the better of his tricky father-in-law. Some animal breeders still profess faith in it as a part of their methods of breeding: if they want a black calf, for instance, they will keep a white cow in a black stall, and express perfect confidence that her offspring will resemble midnight darkness. It is easy to see that this method, if it "works," would be a potent instrument for eugenics. And it is being recommended for that reason. Says a recent writer, who professes on the cover of her book to give a "complete and intelligent summary of all the principles of eugenics":

"Too much emphasis can not be placed upon the necessity of young people making the proper choice of mates in marriage; yet if the production of superior children were dependent upon that one factor, the outlook would be most discouraging to prospective fathers and mothers, for weak traits of character are to be found in all. But when young people learn that by a conscious endeavor to train themselves, they are thereby training their unborn children, they can feel that there is some hope and joy in parentage; that it is something to which they can look forward with delight and even rapture; then they will be inspired to work hard to attain the best and highest that there is in them, leading the lives that will not only be a blessing to themselves, but to their succeeding generation."

The author of this quotation has no difficulty in finding supporters. Many physicians and surgeons, who are supposed to be trained in scientific methods of thought, will indorse what she says. The author of one of the most recent and in many respects admirable books on the care of babies, is almost contemptuous in her disdain for those who think otherwise:

"Science wrangles over the rival importance of heredity and environment, but we women know what effects prenatal influence works on children." "The woman who frets brings forth a nervous child. The woman who rebels generally bears a morbid child." "Self-control, cheerfulness and love for the little life breathing in unison with your own will practically insure you a child of normal physique and nerves."

Such statements, backed up by a great array of writers and speakers whom the layman supposes to be scientific, and who think themselves scientific, can not fail to influence strongly an immense number of fathers and mothers. If they are truly scientific statements, their general acceptance must be a great good.

But think of the misplaced effort if these widespread statements are false!

Is there, or is there not, a short cut to race betterment? Everyone interested in the welfare of the race must feel the necessity of getting at the truth in the case; and the truth can be found only by rigorously scientific thought.

Let us turn to the observed facts. This sample is taken from the health department of a popular magazine, quite recently issued:

"Since birth my body has been covered with scales strikingly resembling the surface of a fish. My parents and I have expended considerable money on remedies and specialists without deriving any permanent benefit. I bathe my entire body with hot water daily, using the best quality of soap. The scales fall off continually. My brother, who is younger than myself, is afflicted with the same trouble, but in a lesser degree. My sister, the third member of the family, has been troubled only on the knees and abdomen. My mother has always been quite nervous and susceptible to any unusual mental impression. She believes that she marked me by craving fish, and preferring to clean them herself. During the prenatal life of my brother, she worried much lest she might mark him in the same way. In the case of my sister she tried to control her mind."[25]

Another is taken from a little publication which is devoted to eugenics.[26] As a "horrible example" the editor gives the case of Jesse Pomeroy, a murderer whom older readers will remember. His father, it appears, worked in a meat market. Before the birth of Jesse, his mother went daily to the shop to carry a luncheon to her husband, and her eyes naturally fell upon the bloody carcases hung about the walls. Inevitably, the sight of such things would produce bloody thoughts in the mind of the unborn child!

These are extreme cases; we quote from a medieval medical writer another case that carries the principle to its logical conclusion: A woman saw a Negro,—at that time a rarity in Europe. She immediately had a sickening suspicion that her child would be born with a black skin. To obviate the danger, she had a happy inspiration—she hastened home and washed her body all over with warm water. When the child appeared, his skin was found to be normally white—except between the fingers and toes, where it was black. His mother had failed to wash herself thoroughly in those places!

Of course, few of the cases now credited are as gross as this, but the principle involved remains the same.

We will take a hypothetical case of a common sort for the sake of clearness: the mother receives a wound on the arm; when her child is born it is found to have a scar of some sort at about the same place on the corresponding arm. Few mothers would fail to see the result of a maternal impression here. But how could this mark have been transmitted? This is not a question of the transmission of acquired characters through the germ-plasm, or anything of that sort, for the child was already formed when the mother was injured. One is obliged, therefore, to believe that the injury was in some way transmitted through the placenta, the only connection between the mother and the unborn child; and that it was then reproduced in some way in the child.

Here is a situation which, examined in the cold light of reason, puts a heavy enough strain on the credulity. Such an influence can reach the embryo only through the blood of the mother. Is it conceivable to any rational human being, that a scar, or what not, on the mother's body can be dissolved in her blood, pass through the placenta into the child's circulation, and then gather itself together into a definite scar on the infant's arm?

There is just as much reason to expect the child to grow to resemble the cow on whose milk it is fed after birth, as to expect it to grow to resemble its mother, because of prenatal influence, as the term is customarily used, for once development has begun, the child draws nothing more than nourishment from its mother.

Of course we are accustomed to the pious rejoinder that man must not expect to understand all the mysteries of life; and to hear vague talk about the wonder of wireless telegraphy. But wireless telegraphy is something very definite and tangible—there is little mystery about it. Waves of a given frequency are sent off, and caught by an instrument attuned to the same frequency. How any rational person can support a belief in maternal impressions by such an analogy, if he knows anything about anatomy and physiology, passes comprehension.

Now we are far from declaring that a reason can be found for everything that happens. Science does not refuse belief in an observed fact merely because it is unexplainable. But let us examine this case of maternal impressions a little further. What can be learned of the time element?

Immediately arises the significant fact that most of the marks, deformities and other effects which are credited to prenatal influence must on this hypothesis take place at a comparatively late period in the antenatal life of the child. The mother is frightened by a dog; the child is born with a dog-face. If it be asked when her fright occurred, it is usually found that it was not earlier than the third month, more likely somewhere near the sixth.

But it ought to be well known that the development of all the main parts of the body has been completed at the end of the second month. At that time, the mother rarely does more than suspect the coming of the child, and events which she believes to "mark" the child, usually occur after the fourth or fifth month, when the child is substantially formed, and it is impossible that many of the effects supposed to occur could actually occur. Indeed, it is now believed that most errors of development, such as lead to the production of great physical defects, are due to some cause within the embryo itself, and that most of them take place in the first three or four weeks, when the mother is by no means likely to influence the course of embryological development by her mental attitude toward it, for the very good reason that she knows nothing about it.

Unless she is immured or isolated from the world, nearly every expectant mother sees many sights of the kind that, according to popular tradition, cause "marks." Why is it that results are so few? Why is it that women doctors and nurses, who are constantly exposed to unpleasant sights, have children that do not differ from those of other mothers?

Darwin, who knew how to think scientifically, saw that this is the logical line of proof or disproof. When Sir Joseph Hooker, the botanist and geologist who was his closest friend, wrote of a supposed case of maternal impression, one of his kinswomen having insisted that a mole which appeared on her child was the effect of fright upon herself for having, before the birth of the child, blotted with sepia a copy of Turner's Liber Studiorum that had been lent her with special injunctions to be careful, Darwin[27] replied: "I should be very much obliged, if at any future or leisure time you could tell me on what you ground your doubtful belief in imagination of a mother affecting her offspring. I have attended to the several statements scattered about, but do not believe in more than accidental coincidences. W. Hunter told my father, then in a lying-in hospital, that in many thousand cases he had asked the mother, before her confinement, whether anything had affected her imagination, and recorded the answers; and absolutely not one case came right, though, when the child was anything remarkable, they afterwards made the cap to fit."

Any doctor who has handled many maternity cases can call to mind instances where every condition was present to perfection, for the production of maternal impression, on the time-honored lines. None occurred. Most mothers can, if they give the matter careful consideration, duplicate this experience from their own. Why is it that results are so rare?

That Darwin gave the true explanation of a great many of the alleged cases is perfectly clear to us. When the child is born with any peculiar characteristic, the mother hunts for some experience in the preceding months that might explain it. If she succeeds in finding any experience of her own at all resembling in its effects the effect which the infant shows, she considers she has proved causation, has established a good case of prenatal influence.

It is not causation; it is coincidence.

If the prospective mother plays or sings a great deal, with the idea of giving her child a musical endowment, and the child actually turns out to have musical talent, the mother at once recalls her yearning that such might be the case; her assiduous practice which she hoped would be of benefit to her child. She immediately decides that it did benefit him, and she becomes a convinced witness to the belief in prenatal culture. Has she not herself demonstrated it?

She has not. But if she would examine the child's heredity, she would probably find a taste for music running in the germ-plasm. Her study and practice had not the slightest effect on this hereditary disposition; it is equally certain that the child would have been born with a taste for music if its mother had devoted eight hours a day for nine months to cultivating thoughts of hatred for the musical profession and repugnance for everything that possesses rhythm or harmony.

It necessarily follows, then, that attempts to influence the inherent nature of the child, physically or mentally, through "prenatal culture," are doomed to disappointment. The child develops along the lines of the potentialities which existed in the two germ-cells that united to become its origin. The course of its development can not be changed in any specific way by any corresponding act or attitude of its mother, good hygiene alone need be her concern.

It must necessarily follow that attempts to improve the race on a large scale, by the general adoption of prenatal culture as an instrument of eugenics, are useless.

Indeed, the logical implication of the teaching is the reverse of eugenic. It would give a woman reason to think she might marry a man whose heredity was most objectionable, and yet, by prenatal culture, save her children from paying the inevitable penalty of this weak heritage. The world has long shuddered over the future of the girl who marries a man to reform him; but think what it means to the future of the race if a superior girl, armed with correspondence school lessons in prenatal culture, marries a man to reform his children!

Those who practice this doctrine are doomed to disillusion. The time they spend on prenatal culture is not cultivating the child; it is merely perpetuating a fallacy. Not only is their time thus spent wasted, but worse, for they might have employed it in ways that really would have benefited the child—in open-air exercise, for instance.

To recapitulate, the facts are:

(1) That there is, before birth, no connection between mother and child, by which impressions on the mother's mind or body could be transmitted to the child's mind or body.

(2) That in most cases the marks or defects whose origin is attributed to maternal impression, must necessarily have been complete long before the incident occurred which the mother, after the child's birth, ascribes as the cause.

(3) That these phenomena usually do not occur when they are, and by hypothesis ought to be, expected. The explanations are found after the event, and that is regarded as causation which is really coincidence.

Pre-natal care as a euthenic measure is of course not only legitimate but urgent. The embryo derives its entire nourishment from the mother; and its development depends wholly on its supply of nourishment. Anything which affects the supply of nourishment will affect the embryo in a general, not a particular way. If the mother's mental and physical condition be good, the supply of nourishment to the embryo is likely to be good, and development will be normal. If, on the other hand, the mother is constantly harassed by fear or hatred, her physical health will suffer, she will be unable properly to nourish her developing offspring, and it may be its poor physical condition when born, indicates this.

Further, if the mother experiences a great mental or physical shock, it may so upset her health that her child is not properly nourished, its development is arrested, mentally as well as physically, and it is born defective. H. H. Goddard, for example, tells[28] of a high-grade imbecile in the Training School at Vineland, N. J. "Nancy belongs to a thoroughly normal, respectable family. There is nothing to account for the condition unless one accepts the mother's theory. While it sounds somewhat like the discarded theory of maternal impression, yet it is not impossible that the fright and shock which the mother received may have interfered with the nutrition of the unborn child and resulted in the mental defect. The story in brief is as follows. Shortly before this child was born, the mother was compelled to take care of a sister-in-law who was in a similar condition and very ill with convulsions. Our child's mother was many times frightened severely as her sister-in-law was quite out of her mind."

It is easily understandable that any event which makes such an impression on the mother as to affect her health, might so disturb the normal functioning of her body that her child would be badly nourished, or even poisoned. Such facts undoubtedly form the basis on which the airy fabric of prenatal culture was reared by those who lived before the days of scientific biology.

Thus, it is easy enough to see the real explanation of such cases as those mentioned near the beginning of this discussion. The mothers who fret and rebel over their maternity, she found, are likely to bear neurotic children. It is obvious (1) that mothers who fret and rebel are quite likely themselves to be neurotic in constitution, and the child naturally gets its heredity from them: (2) that constant fretting and rebellion would so affect the mother's health that her child would not be properly nourished.

When, however, she goes on to draw the inference that "self-control, cheerfulness and love ... will practically insure you a child normal in physique and nerves," we are obliged to stop. We know that what she says is not true. If the child's heredity is bad, neither self-control, cheerfulness, love, nor anything else known to science, can make that heredity good.

At first thought, one may wish it were otherwise. There is something inspiring in the idea of a mother overcoming the effect of heredity by the sheer force of her own will-power. But perhaps in the long run it is as well; for there are advantages on the other side. It should be a satisfaction to mothers to know that their children will not be marked or injured by untoward events in the antenatal days; that if the child's heredity can not be changed for the better, neither can it be changed for the worse.

The prenatal culturists and maternal-impressionists are trying to place on her a responsibility which she need not bear. Obviously, it is the mother who is most nearly concerned with the bogy of maternal impressions, and it should make for her peace of mind to know that it is nothing more than a bogy. It is important for the expectant mother to keep herself in as nearly perfect condition as possible, both physically and mentally. Her bodily mechanism will then run smoothly, and the child will get from her blood the nourishment needed for its development. Beyond that there is nothing the mother can do to influence the development of her child.

There is another and somewhat similar fallacy which deserves a passing word, although it is of more concern to the livestock breeder than to the eugenist. It is called telegony and is, briefly, this: that conception by a female results in a definite modification of her germ-plasm from the influence of the male, and that this modification will be shown in the offspring she may subsequently bear to a second male. The only case where it is often invoked in the human race is in miscegenation. A white woman has been married to a Negro, for instance, and has borne one or more mulatto offspring. Subsequently, she mates with a white man; but her children by him, instead of being pure white, it is alleged, will be also mulattoes. The idea of telegony, the persistent influence of the first mating, may be invoked to explain this discrepancy.

It is a pure myth. There is no good evidence[29] to support it, and there is abundant evidence to contradict it. Telegony is still believed by many animal breeders, but it has no place in science. In such a case as the one quoted, the explanation is undoubtedly that the supposed father is not the real one; and this explanation will dispose of all other cases of telegony which can not be explained, as in most instances they can be, by the mixed ancestry of the offspring and the innate tendency of all living things to vary.

Now to sum up this long chapter. We started with a consideration of the germ-plasm, the physical basis of life; pointing out that it is continuous from generation to generation, and potentially immortal; that it is carefully isolated and guarded in the body, so that it is not likely to be injured by any ordinary means.

One of the logical results of this continuity of the germ-plasm is that modifications of the body of the parent, or acquired characters, can hardly be transferred to the germ-plasm and become a part of the inheritance. Further the experimental evidence upholds this position, and the inheritance of acquired body characters may be disregarded by eugenics, which is therefore obliged to concern itself solely with the material already in existence in the germ-plasm, except as that material may be changed by variation which can neither be predicted nor controlled.

The evidence that the germ-plasm can be permanently modified does not warrant the belief; and such results, if they exist at all, are not large enough or uniform enough to concern the eugenist.

Pre-natal culture and telegony were found to be mere delusions. There is no justification for hoping to influence the race for good through the action of any kind of external influences; and there is not much danger of influencing it for ill through these external influences. The situation must be faced squarely then: if the race is to be improved, it must be by the use of the material already in existence; by endeavor to change the birth-and death-rates so as to alter the relative proportions of the amounts of good and bad germ-plasm in the race. This is the only road by which the goal of eugenics can be reached.



CHAPTER III

DIFFERENCES AMONG MEN

While Mr. Jefferson, when he wrote into the Declaration of Independence his belief in the self-evidence of the truth that all men are created equal, may have been thinking of legal rights merely, he was expressing an opinion common among philosophers of his time. J. J. Rousseau it was who made the idea popular, and it met with widespread acceptance for many years. It is not surprising, therefore, that the phrase has long been a favorite with the demagogue and the utopian. Even now the doctrine is by no means dead. The American educational system is based largely on this dogma, and much of the political system seems to be grounded on it. It can be seen in the tenets of labor unions, in the practice of many philanthropies—traces may be found almost anywhere one turns, in fact.

Common enough as applied to mental qualities, the theory of human equality is even more widely held of "moral" qualities. Men are considered to be equally responsible for their conduct, and failure to conform to the accepted code in this respect brings punishment. It is sometimes conceded that men have had differing opportunities to learn the principles of morality; but given equal opportunities, it is almost universally held that failure to follow the principles indicates not inability but unwillingness. In short, public opinion rarely admits that men may differ in their inherent capacity to act morally.

In view of its almost universal and unquestioned, although half unconscious, acceptance as part of the structure of society, it becomes of the utmost importance that this doctrine of human equality should be examined by scientific methods.

Fortunately this can be done with ease. Methods of mental and physical measurement that have been evolved during the last few decades offer results that admit of no refutation, and they can be applied in hundreds of different places.



It will not be worth while to spend any time demonstrating that all individuals differ, at birth and during their subsequent life, physically. The fact is patent to all. It carries with it as a necessary corollary mental differences, since the brain is part of the body; nevertheless, we shall demonstrate these mental differences independently.

We present in Fig. 8 a graph from E. L. Thorndike, showing the number of 10-year-old children in Connecticut (1903) in each school grade. If the children are all intellectually equal, all the 10-year-olds ought to be in the same grade, or near it. Numerous explanations of their wide distribution suggest themselves; as a working hypothesis one might adopt the suggestion that it is because the children actually differ in innate ability to the extent here indicated. This hypothesis can be tested by a variety of mental measurements. S. A. Courtis' investigation of the arithmetical abilities of the children in the schools of New York City will be a good beginning. He measured the achievements of pupils in responding to eight tests, which were believed to give a fair idea of the pupil's capacity for solving simple arithmetical problems. The results were, on the average, similar to the result he got in a certain eighth-grade class, whose record is shown in Fig. 9. It is evident that some of the children were good in arithmetic, some were poor in it; the bulk of them were neither good nor bad but half way between, or, in statistical language, mediocre.



The literature of experimental psychology and anthropology is crammed with such examples as the above. No matter what trait of the individual be chosen, results are analogous. If one takes the simplest traits, to eliminate the most chances for confusion, one finds the same conditions every time. Whether it be speed in marking off all the A's in a printed sheet of capitals, or in putting together the pieces of a puzzle, or in giving a reaction to some certain stimulus, or in making associations between ideas, or drawing figures, or memory for various things, or giving the opposites of words, or discrimination of lifted weights, or success in any one of hundreds of other mental tests, the conclusion is the same. There are wide differences in the abilities of individuals, no two being alike, either mentally or physically, at birth or any time thereafter.



Whenever a large enough number of individuals is tested, these differences arrange themselves in the same general form. It is the form assumed by the distribution of any differences that are governed absolutely by chance.

Suppose an expert marksman shoots a thousand times at the center of a certain picket in a picket fence, and that there is no wind or any other source of constant error that would distort his aim. In the long run, the greatest number of his shots would be in the picket aimed at, and of his misses there would be just as many on one side as on the other, just as many above as below the center. Now if all the shots, as they struck the fence, could drop into a box below, which had a compartment for each picket, it would be found at the end of his practice that the compartments were filled up unequally, most bullets being in that representing the middle picket and least in the outside ones. The intermediate compartments would have intermediate numbers of bullets. The whole scheme is shown in Fig. 11. If a line be drawn to connect the tops of all the columns of bullets, it will make a rough curve or graph, which represents a typical chance distribution. It will be evident to anyone that the distribution was really governed by "chance," i.e., a multiplicity of causes too complex to permit detailed analysis. The imaginary sharp-shooter was an expert, and he was trying to hit the same spot with each shot. The deviation from the center is bound to be the same on all sides.



Now suppose a series of measurements of a thousand children be taken in, let us say, the ability to do 18 problems in subtraction in 10 minutes. A few of them finish only one problem in that time; a few more do two, more still are able to complete three, and so on up. The great bulk of the children get through from 8 to 12 problems in the allotted time; a few finish the whole task. Now if we make a column for all those who did one problem, another column beside it for all those who did two, and so on up for those who did three, four and on to eighteen, a line drawn over the tops of the columns make a curve like the above from Thorndike.

Comparing this curve with the one formed by the marksman's spent bullets, one can not help being struck by the similarity. If the first represented a distribution governed purely by chance, it is evident that the children's ability seems to be distributed in accordance with a similar law.

With the limited number of categories used in this example, it would not be possible to get a smooth curve, but only a kind of step pyramid. With an increase in the number of categories, the steps become smaller. With a hundred problems to work out, instead of 18, the curve would be something like this:



And with an infinite number, the steps would disappear altogether, leaving a perfectly smooth, flowing line, unmarred by a single step or break. It would be an absolutely continuous distribution.

If then, the results of all the tests that have been made on all mental traits be studied, it will be found that human mental ability as shown in at least 95% of all the traits that have been measured, is distributed throughout the race in various degrees, in accordance with the law of chance, and that if one could measure all the members of the species and plot a curve for these measurements, in any trait, he would get this smooth, continuous curve. In other words, human beings are not sharply divided into classes, but the differences between them shade off into each other, although between the best and the worst, in any respect, there is a great gulf.

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